Study Guide for Campbell Biology
11th Edition
ISBN: 9780134443775
Author: Lisa A. Urry, Michael L. Cain, Steven A. Wasserman, Peter V. Minorsky, Jane B. Reece, Martha R. Taylor, Michael A. Pollock
Publisher: PEARSON
expand_more
expand_more
format_list_bulleted
Concept explainers
Question
Chapter 18, Problem 23TYK
Summary Introduction
Introduction: Apoptosis is also called programmed cell death. It involves biochemical mechanisms that lead to cell death. The apoptosis helps to prevent the development of cancer. The apoptosis is initiated by various factors in response to internal and external factors.
Expert Solution & Answer
Want to see the full answer?
Check out a sample textbook solutionStudents have asked these similar questions
Mutations to proto-oncogenes act toa. increase cell division. c. repress oncogenes.b. increase DNA repair. d. decrease cell division
Hypermethylation is thought to contribute to cancer by
a. inhibiting DNA replication.
b. inhibiting the expression of tumor-suppressor genes.
c. stimulating the translation of oncogenes.
d. stimulating telomerase.
Which of the following statements
concerning p53 is NOT correct?
O a. p53-dependent transcription of target genes leads
to cell cycle arrest or cell death (apoptosis).
O b.
O C.
Loss of p53 function results in loss of checkpoint
controls.
Loss of p53 function creates an environment that
is permissive for genome instability--that is, more
damaged cells with chromosome aberrations and
mutations survive and propagate.
d. p53 is a tumor suppressor protein which is largely
responsible for protecting cells from cancer-
causing DNA-damaging agents.
Chapter 18 Solutions
Study Guide for Campbell Biology
Ch. 18 - In the following diagram of the lac operon, an...Ch. 18 - a. Repressible enzymes usually function in _____...Ch. 18 - a. Give an example of highly methylated and...Ch. 18 - Label the components of the following diagram that...Ch. 18 - a. How is the process of alternate RNA splicing...Ch. 18 - a. Describe how miRNAs regulate gene expression....Ch. 18 - a. What is the difference between determination...Ch. 18 - What type of evidence established that Bicoid...Ch. 18 - a. List three genetic changes that can convert a...Ch. 18 - Complete the following concept map to help you...
Ch. 18 - Fill in the following table to help you organize...Ch. 18 - Prob. 3SYKCh. 18 - Prob. 1TYKCh. 18 - Prob. 2TYKCh. 18 - Prob. 3TYKCh. 18 - Prob. 4TYKCh. 18 - Prob. 5TYKCh. 18 - DNA methylation of cytosine bases a. initiates the...Ch. 18 - Which of the following is not true of enhancers?...Ch. 18 - Prob. 8TYKCh. 18 - A eukaryotic gene typically has all of the...Ch. 18 - Prob. 10TYKCh. 18 - Prob. 11TYKCh. 18 - Which of the following statements explains why a...Ch. 18 - Prob. 13TYKCh. 18 - Prob. 14TYKCh. 18 - Prob. 15TYKCh. 18 - Prob. 16TYKCh. 18 - What would be the fate of a Drosophila larva that...Ch. 18 - Prob. 18TYKCh. 18 - Prob. 19TYKCh. 18 - Prob. 20TYKCh. 18 - Prob. 21TYKCh. 18 - Prob. 22TYKCh. 18 - Prob. 23TYKCh. 18 - Which of the following would most likely account...
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Assuming that apoptosis fails and the cell continues through several cell cycles... what would the repercussions of this be? A. a tumor (possibly malignant) may develop- result of cells actively dividing when they are not supposed to B. organ failure C. too many cells would be in G0 and not carrying out daily functions D. too many mutated cells would be in the body and the body would shut downarrow_forwardSometimes, malignant cells do not stay properly anchored in tissues because of the loss of function of: a. cell walls b. oncogenes c. plasma membrane adhesion proteins d. nuclear membranes e. tumor suppressor genesarrow_forwardNormal (nonmutant) tumor-suppressor genes often functiona. as negative regulators of cell division.b. in the maintenance of genome integrity.c. in the stimulation of cell division.d. as both a and b.arrow_forward
- Which of the following demonstrates the link between oncogenes and cancer? a.Oncogenes do not have mutations that increase the activity or number of molecules that stimulate mitosis. b.Oncogenes produce molecules that inhibit mitosis. c.They are genes that transform tumor cells into normal cells. d.The mutations in oncogenes increase the activity or number of molecules that stimulate mitosis, leading to irregular cell division.arrow_forwardWhich of the following types of mutations would be advantageous to a cancer cell (select all that apply)? A. An inactivating mutation in a tumor suppressor gene B. Methylation of the promoter of a tumor suppressor gene C. An inactivating mutation in an oncogene D. Mutation that inactivated DNA repair gene E. An inactivating mutation in an oncogenearrow_forwardMost forms of cancer involvea. the activation of a single oncogene.b. the inactivation of a single tumor-suppressor gene.c. the activation of multiple oncogenes.d. the activation of multiple oncogenes and the inactivation ofmultiple tumor-suppressor genes.arrow_forward
- Which of the following is typical of cancer cells? A. The parent cell of the tumor contains a single mutation in a single checkpoint gene. B. Malignant cells migrate. C. Cancer cells lose the ability to divide. D. Products of oncogenes inhibit mitosis E. Benign tumors invade normal tissue.arrow_forwardA tumor suppressor genea. inhibits cell division.b. opposes oncogenes.c. prevents cancer.d. is subject to mutations.e. All of these are correct.arrow_forwardWhich of the following is NOT a way in which proto-oncogenes can change to become genes that induce cancer? Group of answer choices a. changes in a control element (enhancer) to increase transcription b. gene amplification c. changes in DNA sequence to produce a product that degrades rapidly d. movement of the gene adjacent to a different control element to increase transcription e. changes in DNA sequence to produce a product that isarrow_forward
- Cancer Cells need A.I.R in order to survive and proliferate. What does this stand for? a. Activation of TSG's; Inactivation of oncogene; Replenishing of Telomeres b. Absorption of oncogenes; Inactivation of TRK's; Replenishing of Telomeres c. Activation of oncogenes; Inactivation of TGF's; Replenishing of Telomeres d. Activation of oncogene; Inactivation of TSG's; Replenishing of Telomeresarrow_forwardWhich of the following accurately compares tumor suppressor genes and proto-oncogenes? A. Both tumor suppressor and proto-oncogenes prevent the spread of cancer by targeting and destroying existing cancer cells. B. A person who has both tumor suppressor and proto-oncogenes will always develop cancer. C. Proto-oncogenes code for growth factors, while tumor suppressor genes inhibit cell division of damaged cells. D. When a proto-oncogene mutates, it becomes a tumor suppressor gene.arrow_forwardIdentify which of the following statements is a lie? Select one: a. Apoptosis occurs as the body produces certain enzymes that initiate degradation of nuclear and cytoplasmic material, breaking the cell into necrosis bodies. b. Apoptosis helps maintain a healthy and normal population of cells. c. Apoptosis plays an essential role in growth, immune surveillance, and embryological development.arrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you
- Concepts of BiologyBiologyISBN:9781938168116Author:Samantha Fowler, Rebecca Roush, James WisePublisher:OpenStax College
Concepts of Biology
Biology
ISBN:9781938168116
Author:Samantha Fowler, Rebecca Roush, James Wise
Publisher:OpenStax College
Mitochondrial mutations; Author: Useful Genetics;https://www.youtube.com/watch?v=GvgXe-3RJeU;License: CC-BY