Human Heredity: Principles and Issues (MindTap Course List)
11th Edition
ISBN: 9781305251052
Author: Michael Cummings
Publisher: Cengage Learning
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Chapter 7, Problem 18QP
Equalizing the Expression of X Chromosome Genes in Males and Females
Males have only one X chromosome and therefore only one copy of all genes on the X chromosome. Each gene is directly expressed, thus providing the basis of hemizygosity in males. Females have two X chromosomes, but one is always inactivated. Therefore, females, like males, have only one functional copy of all the genes on the X chromosome. Again, each gene must be directly expressed. Why, then, are females not considered hemizygous, and why are they not afflicted with sex-linked recessive diseases as often as males are?
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Chapter 7 Solutions
Human Heredity: Principles and Issues (MindTap Course List)
Ch. 7.6 - Prob. 1EGCh. 7.6 - Prob. 2EGCh. 7 - As outlined in this chapter, sex can be defined at...Ch. 7 - As outlined in this chapter, sex can be defined at...Ch. 7 - Prob. 1QPCh. 7 - The Human Reproductive System Discuss and compare...Ch. 7 - Prob. 3QPCh. 7 - A Survey of Human Development from Fertilization...Ch. 7 - Prob. 5QPCh. 7 - Prob. 6QP
Ch. 7 - Prob. 7QPCh. 7 - How Is Sex Determined? The absence of a Y...Ch. 7 - Prob. 9QPCh. 7 - Mutations Can Uncouple Chromosomal Sex from...Ch. 7 - Prob. 11QPCh. 7 - Mutations Can Uncouple chromosomal Sex from...Ch. 7 - Prob. 13QPCh. 7 - Sex-Influenced and Sex-Limited Traits What method...Ch. 7 - Prob. 15QPCh. 7 - Prob. 16QPCh. 7 - Equalizing the Expression of X Chromosome Genes in...Ch. 7 - Equalizing the Expression of X Chromosome Genes in...Ch. 7 - Equalizing the Expression of X Chromosome Genes in...
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- Equalizing the Expression of X Chromosome Genes in Males and Females Individuals with an XXY genotype are sterile males. If one X is inactivated early in embryogenesis, the genotype of the individual effectively becomes XY. Why will this individual not develop as a normal male?arrow_forwardThe expression patterns as well as activation of different types of CDKs happen at different stages of the cell cycle. Explain why.arrow_forwardThe P63 and P53 have similar functionalities in the cell, however, p53 is rarely associated directly with p63, suggesting that p63 may indirectly act as an oncogene by blocking p53 function. This hypothesis may also explain why p63 is associated with other indications of misinterpretation. I do understand the above statement, however once piece not clear – why would p63 block p53 function? Have these genes been shown to have opposing functions? From the background information provided above, it seems like they would have seminar functions. Explain.arrow_forward
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