Study Guide for Campbell Biology
11th Edition
ISBN: 9780134443775
Author: Lisa A. Urry, Michael L. Cain, Steven A. Wasserman, Peter V. Minorsky, Jane B. Reece, Martha R. Taylor, Michael A. Pollock
Publisher: PEARSON
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Chapter 48, Problem 13TYK
Summary Introduction
Introduction: The cells of the nervous system that control the transmission of signals are called neurons. The neurons have several types of voltage-gated ions channels. Sodium and potassium are important ions that regulate the opening and closing of these channels.
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Neurotransmitters:
a. Can only inhibit the postsynaptic cell
b. Can be part of a process that regulates transcription and translation in the postsynaptic cell
с.
Are bound to their receptors forever, until the receptor is degraded and recycled.
d. Can exit the presynaptic cell without assistance.
e. Can only excite the postsynaptic cell
Clathrin:
Is embedded in the cell membrane of the synaptic terminal.
a.
b. Degrades vesicles for recycling and return to the axon hillock
с.
Mediates ALL endocytosis in the body, including the formation of vesicles in the synapse.
d. Forms a coat of octagonal proteins.
e. Helps the vesicle leave the pre-synaptic cell, and fuse to the membrane of the post-synaptic
cell
Both potassium and sodium channels located along the nerve membrane are voltage-gated. This means that they respond to the changes in the voltage by opening and closing the gates to allow the ions to flow in or out of the membrane.
When the threshold level is reached and depolarization occurs, how come only the sodium ions begin to rush in?
a. The threshold levels for both sodium and potassium channels are equal, but sodium channels are slower to open.
b. The threshold levels for both sodium and potassium channels are equal, but potassium channels take longer to open.
c. The threshold level for the potassium channels is lower than the sodium channels.
d. The threshold level for the potassium channels is higher than the sodium channels.
Which of the following is true about the conduction of action potentials?
a. Thicker axons are faster because there is more surface area on thicker axons.
b. Myelin speeds conduction because the glial cells add voltage gated Na+ channels to the neuron.
c. Diffusion of Na+ ions between the Nodes of Ranvier happens faster than the wave of opening and closing membrane proteins can travel.
d. Neurons that don’t have myelin sheaths undergo saltatory conduction
e. Action potentials move slowly.
Chapter 48 Solutions
Study Guide for Campbell Biology
Ch. 48 - Prob. 1IQCh. 48 - a. What is the principal cation inside the cell?...Ch. 48 - The following diagram shows the changes in...Ch. 48 - Prob. 4IQCh. 48 - Prob. 5IQCh. 48 - Prob. 6IQCh. 48 - Prob. 7IQCh. 48 - Develop a flowchart or diagram or write a...Ch. 48 - Prob. 1TYKCh. 48 - Prob. 2TYK
Ch. 48 - During a neurons resting state a. there are more...Ch. 48 - Which of the following contribute(s) to the...Ch. 48 - Prob. 5TYKCh. 48 - Prob. 6TYKCh. 48 - Prob. 7TYKCh. 48 - After the rapid depolarization of an action...Ch. 48 - Nodes of Ranvier are a. gaps where Schwann cells...Ch. 48 - Prob. 10TYKCh. 48 - Signal transmission is faster in myelinated axons...Ch. 48 - Which of the following statements concerning...Ch. 48 - Prob. 13TYKCh. 48 - Prob. 14TYKCh. 48 - Prob. 15TYKCh. 48 - Prob. 16TYKCh. 48 - Prob. 17TYKCh. 48 - If the binding of a neurotransmitter to its...
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- What does a transporter protein do at a synapse? a. It carries neurotransmitter molecules from the presynaptic neuron to the postsynaptic neuron. b. It carries neurotransmitter molecules from the cell body to the presynaptic terminal. c. It carries neurotransmitter molecules from neurons that have too much into neurons that need more. d. It pumps used neurotransmitter molecules back into the presynaptic neuron.arrow_forwardA neuron must reach threshold to fire an action potential. In this context, threshold refers to which event? a. voltage at which damage occurs to the membrane because of the electrical charge b. the voltage at which potassium ions begin to flow in and out of the cell c. the voltage at which voltage-gated sodium channels open d. the voltage at which the neuron can no longer produce an action potential According to studies in developing rodents, testosterone treatment causes detectable changes in: a. the sex region Y gene b. the thalamus c. the hypothalamus d. alpha-fetoproteinarrow_forwardThe nerve impulses always travel in one direction from dendrites to axon terminals. What prevents an action potential from travelling backward? a. Hyperpolarization of the neuron makes the membrane more positive and prevents another action potential. b. After repolarization, the refractory period occurs, where the sodium ions are found outside the cell. c. After repolarization, the reversal of ions prevents another action potential as there would not be enough sodium ions outside the cell to rush in. d. The refractory period prevents another action potential by lowering the threshold potential.arrow_forward
- What membrane protein of the neuron is responsible for briefly increasing the Na+ permeability of the membrane during the rising phase of the nerve impulse? a. the sodium-potassium pump b. the voltage gated potassium channel c. the voltage gated sodium channel d. the stimulus gated sodium channel e. the ligand gated ion channelarrow_forwardThe axon hillock of a neuron reaches the threshold of excitation and the membrane potential becomes completely reversed. Then, suddenly, the membrane potential begins to return to resting membrane potential. What is the event that triggers this return to resting potential? A. Na+ channels close in a voltage-dependent manner B. Na+ channels close in a time-dependent manner C. Na+ channels close in a chemically-dependent manner D. Na+ channels open in a voltage-dependent mannerarrow_forwardWhich one of the following statements about gap junctions or electrical synapses is incorrect? A. Abundant gap junctions between cardiac muscle cells allow coordinated waves of contraction to spread through the heart muscle. B. Synaptic delays at electrical synapses are shorter than synaptic delays at chemical synapses. C. Gap junctions at electrical synapses are formed from proteins made in both the presynaptic and postsynaptic cells. D. Electrical synapses can be either excitatory or inhibitory. E. Electrical synapses are inherently bidirectional, i.e., current can spread between two coupled cells in either direction Please give me the correct answer quickly I will give you upvotearrow_forward
- Sequence the following list of events of a neuronal action potential by placing 1 next to the first event, 2 next to the second event, and so on. a. _____ The activation gates of voltage-gated Na+ channels open, Na+ flood the cytoplasm, and depolarization occurs. b. _____ K+ continue to flow out of the axon until the membrane is hyperpolarized. c. _____ Local potentials cause the membrane to depolarize to threshold. d. _____ The inactivation gates of voltage-gated Na+ channels close as voltage-gated K+ channels open, K+ begin to exit the axon, and repolarization begins. e. _____ Repolarization continues and Na+ channels return to restingarrow_forwardMany neurotoxins have been used extensively in physiological studies of neurons. When comparing tetrodotoxin found in puffer fish and batrachotoxins from poison dart-frogs, the former had no effect on a neurons resting potential but completely stopped its action potential, whereas the later toxin immediately initiates depolarization of a neuron but prevents repolarization. Can your group hypothesize how these toxins affect the nerve transmission differently?arrow_forwardUse this illustration to place these events in the order they occur during synaptic communication: Column A Column B a. Voltage gated sodium channels carry the action potential to the synapse by allowing the influx of sodium ions. 1. 1 2 b. An increase in calcium levels triggers vesicle release. 3. 3 c. Sodium enters the post-synaptic cell, causing it to depolarize. 4. 4 d. Sodium influx at the synapse activates voltage gated 5. calcium channels which allow calcium ions to enter the presynaptic neuron. 6. 6. e. Neurotransmitters are released into the synaptic cleft. f. Ligand-gated ion channels are opened by the neurotransmitters. 2.arrow_forward
- When an action potential arrives in an axon terminal, synaptic vesicles containing acetylcholine are released by: a. facilitated diffusion. b. osmosis. c. exocytosis. d. endocytosis.arrow_forwardThe figure below may help in answering some of the questions. 1. What ions are flowing (and in which direction) both during the rising phase and the falling phase of the action potential? 2. What are the values of both the 'y' (vertical) axis and the 'x' (horizontal) axis. 3. If extracellular levels of K+ rose (a condition called hyperkalemia), how would that affect the resting membrane potential? 3arrow_forwardIn an experiment you place a neuron and its synaptic contacts into a medium containing no calcium ions. You stimulate the neuron causing an action potential to propagate down the axon into the axon terminal of the neuron. 3.) What is the most likely result of this experimental manipulation? A. Prolong the refractory period of the action potential B. Decrease the time required to move sodium ions out of the axon terminal C. Prevent neurotransmitter from binding to receptors on postsynaptic cells D. Enhance the voltage changes associated with the action potential E. Prevent release of neurotransmitter into the synapsearrow_forward
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