Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN: 9780134580999
Author: Elaine N. Marieb, Katja N. Hoehn
Publisher: PEARSON
expand_more
expand_more
format_list_bulleted
Concept explainers
Question
Sequence the following list of events of a neuronal action potential by placing 1 next to the first event, 2 next to the second event, and so on.
a. _____ The activation gates of voltage-gated Na+ channels open, Na+ flood the cytoplasm, and depolarization occurs.
b. _____ K+ continue to flow out of the axon until the membrane is hyperpolarized.
c. _____ Local potentials cause the membrane to depolarize to threshold.
d. _____ The inactivation gates of voltage-gated Na+ channels close as voltage-gated K+ channels open, K+ begin to exit the axon, and repolarization begins.
e. _____ Repolarization continues and Na+ channels return to resting
Expert Solution
This question has been solved!
Explore an expertly crafted, step-by-step solution for a thorough understanding of key concepts.
This is a popular solution
Trending nowThis is a popular solution!
Step by stepSolved in 2 steps
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Neuron 1 fires an action potential, releasing neurotransmitters to neuron 2. However, neuron 2 does not fire an action potential itself. Use your understanding of neuronal signaling to identify and explain two possible reasons for this observation.arrow_forward13) Draw a graph representing the changes in membrane potential across the axonal membrane before, during, and after an action potential. On your graph, use a highlighter to clearly indicate for which portions of your graph the letter corresponding to each cellular event listed below belongs. List of cellular events: A) All K* channels open B) All Na* channels close C) The Na*/K* ATPase cotransporter transports Na* and K* across the membrane. D) All Na* channels open E) The threshold value of membrane potential is attained. F) All K* channels close G) Some Na* channels open Your graph:arrow_forwardA theoretical drug blocks Na* leak channels - what effect would this have on a neuron's resting membrane potential and why? Would this drug affect the neuron's ability to generate an action potential and why/why not?arrow_forward
- The membrane potential labeled (0) is due to which of the following (when compared to (3) potential. (A) voltage-gated Na+ channels (B) voltage-gated K+ channels (C) voltage-gated slow Ca++ channels (D) voltage-gated fast Ca++ channels (E) voltage-gated transient Ca++ channels (F) funny channelsarrow_forwardWhat type of synaptic potential (if any; be sure to indicate if any modification is occurring as well) would occur if: a.) An MAO inhibitor type of antidepressant is functioning at an active dopaminergic synapse that increases the rate of sodium flowing into the cell. b.) Prozac is present at an active serotonin synapse where receptor activation increases the flow of potassium out of the cell.arrow_forwardPlease answer question 2arrow_forward
- A neuron has a resting membrane potential of -70 milivolts (mV) and a threshold value of -55 mV. Three synapses on the body of this neuron receive the impulses listed below. hyperpolarisation by 5 mV • depolarisation by 15 mV • depolarisation by 10 mV ENTER THE MEMBRANE POTENTIAL AS A NUMBER WITH + OR - IN FRONT OF IT. The final membrane potential will be mV and this cause an action potential becasue it is v than thearrow_forwardWhat statement about synapse activity is false? A.The input of sodium ions by AMPA receptors is triggered by glutamate binding to the same receptors.B.Calcium entering the dendritic spine during synaptic transmission activates proteins that can potentiate synapse.C.Depolarization of the membrane caused by the arrival of calcium ions makes it possible to eject sodium blocking NMDA receptors.D.Glutamate can be released by the pre-synaptic button following the arrival of an action potential via the axon.arrow_forwardNerve membrane hyperpolarization after an action potential a)Is the movement of membrane potential voltage below normal resting potential voltage. b) This Is mostly due to the slow-closing of K+ channels. c)Makes it more difficult to evoke another action potential. d) This Is mostly due to the slow-closing of Na+ channels.arrow_forward
- Conotoxin is produced by marine cone snails. Among its effects is to block voltage-gated Ca2+ channels in n eurons. A. What anatomical part of a neuron would be affected by conotoxin? B. How would the neuron's action potential be affected by conotoxin? Explain, using at least TWO of the following terms: threshold, depolarization, repolarization, hyperpolarization, summation, IPSP, EPSP, exocytosis C. If conotoxin affected a somatic motor neuron, would this toxin cause muscle weakness or increased muscle tension? Explain why.arrow_forwardPlease answer this multiple choice question?arrow_forwardPost synaptic neurons membrane potentials are less negative than resting membrane potentials when this occur A. Creates a new set point for the resting membrane potential B. Has a lower propensity to produce an action potential C. Reacts by generating action potential D. Higher chance of reaching the potential thresholdarrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you
- Human Anatomy & Physiology (11th Edition)BiologyISBN:9780134580999Author:Elaine N. Marieb, Katja N. HoehnPublisher:PEARSONBiology 2eBiologyISBN:9781947172517Author:Matthew Douglas, Jung Choi, Mary Ann ClarkPublisher:OpenStaxAnatomy & PhysiologyBiologyISBN:9781259398629Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa StouterPublisher:Mcgraw Hill Education,
- Molecular Biology of the Cell (Sixth Edition)BiologyISBN:9780815344322Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter WalterPublisher:W. W. Norton & CompanyLaboratory Manual For Human Anatomy & PhysiologyBiologyISBN:9781260159363Author:Martin, Terry R., Prentice-craver, CynthiaPublisher:McGraw-Hill Publishing Co.Inquiry Into Life (16th Edition)BiologyISBN:9781260231700Author:Sylvia S. Mader, Michael WindelspechtPublisher:McGraw Hill Education
Human Anatomy & Physiology (11th Edition)
Biology
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:PEARSON
Biology 2e
Biology
ISBN:9781947172517
Author:Matthew Douglas, Jung Choi, Mary Ann Clark
Publisher:OpenStax
Anatomy & Physiology
Biology
ISBN:9781259398629
Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa Stouter
Publisher:Mcgraw Hill Education,
Molecular Biology of the Cell (Sixth Edition)
Biology
ISBN:9780815344322
Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter Walter
Publisher:W. W. Norton & Company
Laboratory Manual For Human Anatomy & Physiology
Biology
ISBN:9781260159363
Author:Martin, Terry R., Prentice-craver, Cynthia
Publisher:McGraw-Hill Publishing Co.
Inquiry Into Life (16th Edition)
Biology
ISBN:9781260231700
Author:Sylvia S. Mader, Michael Windelspecht
Publisher:McGraw Hill Education