Neuroactive drugs, such as the antidepressant fluoxetine, function by affecting the activity of neurotransmitters in the brain. After the neurotransmitter serotonin has crossed the synapse and attached to the postsynaptic neuron, it is normally taken back up by vesicles in the presynaptic neuron. Fluoxetine inhibits this reuptake process, causing serotonin to remain in the synapse for longer than usual. A Synapse W. Nerve impulse 10) Which process labelled in the diagram above is inhibited by the action of fluoxetine? a) Process W b) Process X c) Process Y d) Process Z

One medication used to manage the symptoms of Alzheimer's disease is a cholinesterase inhibitor. The medication helps to improve brain functioning and memory.     1.What is the function of cholinesterase in the synapse?    2. How does a cholinesterase inhibitor work in a synapse?    3. Explain the function of cholinesterase inhibitor in increasing brain functioning and memory.  Please include the URL of the website that you used to answer these questions.

Identify the inhibitory neurotransmitter which opens chloride ion channels. gamma amino butyric acid acetylcholine tubocurarine strychnine glutamate Spastic paralysis (resulting from the failure of muscles to relax), induced by the neurotoxin strychnine, occurs when this poison blocks the binding of: the excitatory neurotransmitter acetylcholine to post-synaptic sodium channels the excitatory neurotransmitter glutamate to post-synaptic calcium channels the excitatory neurotransmitter serotonin to post-synaptic potassium channels the inhibitory neurotransmitter glycine to post-synaptic chloride channels the inhibitory neurotransmitter melatonin to post-synaptic bicarbonate channels

Please answer question 37 Please select the most correct option

Please answer question 41

Match the events listed with their order in the mechanisms behind the firing of an action potential under normal conditions (without tetrodotoxin present in the man's body).  The cell has a resting membrane potential of -65 mV. You should match these events at the initial segment of a bipolar neuron. Please note that this is a sequence question. Although not an option below, you should consider that neurotransmitters are bound to ligand-gated receptors causing EPSPs. Now, put the process in the correct sequence: **arrange from 1-10

87.

There are a broad range of anti-epileptic medications currently on the market, with different therapies prescribed for different types and severities of the condition. Given what you have learned about synaptic transmission, which of the following could be a potential therapeutic approach to prevent the spreading of neuronal excitation? (3 correct answers, select all that apply) O A voltage-gated calcium channel blocker/inhibitor selective to glutamate-releasing neurons. O A voltage-gated calcium channel blocker/inhibitor selective to GABA-releasing neurons. O A glutamate receptor blocker (antagonist). O A GABA receptor antagonist. O A glutamate reuptake inhibitor. O A GABA reuptake inhibitor.

Describe the neurotransmitters shown in Section 11.14 in the selected neurotransmitters table. Give examples of how they might occur in the body.

Organize the following steps into the correct signal transmission across a synapse.   1. Neurotransmitter molecules diffuse across the synaptic cleft 2. Neurotransmitter molecules bind to postsynaptic neurotransmitter receptors 3. Synaptic vesicles release neurotransmitter molecules 4. Impulse reaches the synaptic knob of the presynaptic neuron