GABA is an example of an inhibitory neurotransmitter. Drugs that increase the level of GABA are used to treat diseases such as epilepsy where the signal transmission needs to be impaired. The effect of the inhibitory neurotransmitter on the postsynaptic membrane is to: Select one: a. decrease the threshold level b. hyperpolarize the postsynaptic membrane c. depolarize the postsynaptic membrane d. maintain the resting potential
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GABA is an example of an inhibitory neurotransmitter. Drugs that increase the level of GABA are used to treat diseases such as epilepsy where the signal transmission needs to be impaired.
The effect of the inhibitory neurotransmitter on the postsynaptic membrane is to:
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- A postsynaptic neuron has three presynaptic inputs from neurons X, Y, and Z. When X and Y are stimulated simultaneously and repeatedly, the postsynaptic neuron reaches threshold and undergoes an action potential. When X and Z are stimulated simultaneously, however, there is no change in the membrane potential of the postsynaptic neuron. Which of the following is correct? Select one: A. The simultaneous stimulation of X and Z is an example of temporal summation. O B. The simultaneous stimulation of X and Y is an example of temporal summation. O C. The repeated rapid stimulation of X is an example of spatial summation. O D. The stimulation of Y causes an IPSP in the postsynaptic neuron. O E. The stimulation of Z causes a hyperpolarization in the postsynaptic neuron.Postsynaptic potentials are graded potentials that develop in the postsynaptic membrane. Which one of the following statements accurately describes what causes EPSP's to occur at the postsynaptic membrane? Group of answer choices A.an EPSP results from the opening of voltage gated sodium channels B.an EPSP results from the opening of chemically gated sodium channels C.an EPSP results from the opening of voltage gated potassium channels D.an EPSP results from the opening of chemically gated potassium channelsYou apply a drug to a batch of neurons and record membrane potential changes in the presynaptic and postsynaptic neurons. You record an action potential in the presynaptic neuron and a change from -70mV to -55mV in the postsynaptic neuron. Your drug most likely allowed which ion into the postsynaptic neuron? Sodium (Na+) Glutamate Potassium (K+) GABA Chloride (Cl-)
- Depression is a condition in which sufferers may feel low mood, lack of motivation and sleep. It is caused by a lack of serotonin in the synaptic cleft of neurone pathways. Serotonin is a neurotransmitter. a) Describe how serotonin acts as a neurotransmitter. Using your understanding of synaptic transmission, explain how communication between neurones in serotonin pathways of the brain would be affected in someone with depression. b) Doctors use tubocurarine drug as an anaesthetic as it temporarily paralyses muscles. It blocks receptors at neuromuscular junctions. Why does this lead to paralysis?Glutamate's effect when it binds with protein receptors on the post synaptic membrane is to: a. Initiate movement of potassium ions out of the membrane b. Initiate movement of sodium ions into the membrane c. Initiate movement of sodium ions out of the membrane d. Initiate movement of potassium ions into the membrane GABA is an example of an inhibitory neurotransmitter. Drugs that increase the level of GABA are used to treat diseases such as epilepsy where the signal transmission needs to be impaired. The effect of the inhibitory neurotransmitter on the postsynaptic membrane is to: a. hyperpolarize the postsynaptic membrane b. decrease the threshold level c. depolarize the postsynaptic membrane d. maintain the resting potential Calcium is vital for bone growth, but it's also an important part of neural transmission. Calcium is required for the axon terminal to: a. Reuptake the neurotransmitter via exocytosis b. The neurotransmitter to be released by endocytosis c. The neurotransmitter…Name three ways to stop the effect of a neurotransmitteron the postsynaptic membrane. Give an example of each.
- There is a type of toxin found in the liver of the puffer fish that prevents the opening of voltage-gated Na+ channels in neurons. The effect of this toxin is to: Group of answer choices A. prevents the initiation of the action potential B. depolarizes the membrane potential and maintains it depolarized C. prolongs the return of the membrane potential to the resting level D. increases the duration of the action potentialYou apply a drug to a batch of neurons and record membrane potential changes in the presynaptic and postsynaptic neurons. You record an action potential in the presynaptic neuron and a change from -70mV to -85mV in the postsynaptic neuron. Your drug most likely allowed which ion into the postsynaptic neuron? Chloride (Cl-) Sodium (Na+) GABA Potassium (K+) GlutamateAll of the following statements correctly describe a polarized membrane EXCEPT which? a. There is a separation of the charge by a membrane, resulting in its ability to do work. b. An RMP of –60 mV indicates that there are more positive ions inside the axon than an RMP of –70 mV. c. A resting membrane potential (RMP) of –90 mV indicates the there are more positive ions inside the axon than an RMP of –70 mV. d. There is a difference in the charges found on the inside of the membrane to the outside.
- Which of the following are the likely explanations for the failure of a postsynaptic neuron to trigger an action potential? Select all that apply. The EPSPs are neutralized by the neurotransmitter The summation of IPSPs and EPSPs is insufficient to depolarize the cell to threshold The synapses generating the EPSPs are too close to the axon hillock The synapses generating the EPSPs are too far from the axon hillockExplain what happens when an action potential reaches the axon terminus (terminal) by activating the receptors on the postsynaptic membrane. Explanation addressing the question above When an action potential, or nerve impulse, arrives at the axon terminal, it activates voltage-gated calcium channels in the cell membrane. Which is present at a much higher concentration outside the neuron than inside, rushes into the cell. The Ca2+ allows synaptic vesicles to fuse with the axon terminal membrane, releasing neurotransmitters into the synaptic cleft. The molecules of neurotransmitters diffuse across the synaptic cleft and bind to receptor proteins on the postsynaptic cell. Activation of postsynaptic receptors leads to the opening or closing of ion channels in the cell membrane. This may be depolarizing, making the inside of the cell more positive, or hyperpolarizing making the inside more negative depending on the ions involved. The excitatory and inhibitory postsynaptic potentials…Acetylcholine is a neurotransmitter that, when bound to its receptor, causes the receptor to open a channel that allows the flow of Na+ ions into neurons. When the Na+ ions begin to flow into the neuron, this change in the net charge across the neuronal cell membrane often triggers Ca2+ ion channels to open. In this scenario, the acetylcholine receptor would be acting as a channel, and the Ca2+ channels would be classified as voltage-gated; leaky ligand gated; voltage-gated symporter; antiporter None of the above. ion