Biochemistry
9th Edition
ISBN: 9781319114671
Author: Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.
Publisher: W. H. Freeman
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Question
Chapter 27, Problem 4P
Interpretation Introduction
Interpretation:
The pathways that are not stimulated by epinephrine should be determined.
Concept introduction:
Epinephrine, also termed as adrenaline, is a neurotransmitter and hormone that is secreted by the medulla region of the adrenal glands. Anger results in the production of adrenaline into the blood, which causes an increased heart rate, muscle strength, sugar
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Choose the correct. The major action of insulin is: A. Conversion of glucose to glycogenB. ProteolysisC. Conversion of fatty acids to glucoseD. GlycogenolysisE. Gluconeogenesis
Endocrine System Overview
An example of a target cell for glucagon is
a. kidney cells
b. heart cells
c. liver cells
d. adipose cells
. Which of the following pathways is not stimulated by epinephrine? a. Glycogen degradation b. Mobilization of fatty acids from adipose tissue c. Glycogen synthesis d. Glycolysis in muscle
Chapter 27 Solutions
Biochemistry
Ch. 27 - Prob. 1PCh. 27 - Prob. 2PCh. 27 - Prob. 3PCh. 27 - Prob. 4PCh. 27 - Prob. 5PCh. 27 - Prob. 6PCh. 27 - Prob. 7PCh. 27 - Prob. 8PCh. 27 - Prob. 9PCh. 27 - Prob. 10P
Ch. 27 - Prob. 11PCh. 27 - Prob. 12PCh. 27 - Prob. 13PCh. 27 - Prob. 14PCh. 27 - Prob. 15PCh. 27 - Prob. 16PCh. 27 - Prob. 17PCh. 27 - Prob. 18PCh. 27 - Prob. 19PCh. 27 - Prob. 20PCh. 27 - Prob. 21PCh. 27 - Prob. 22PCh. 27 - Prob. 23PCh. 27 - Prob. 24PCh. 27 - Prob. 25PCh. 27 - Prob. 26PCh. 27 - Prob. 27PCh. 27 - Prob. 28PCh. 27 - Prob. 29PCh. 27 - Prob. 30PCh. 27 - Prob. 31PCh. 27 - Prob. 32PCh. 27 - Prob. 33PCh. 27 - Prob. 34PCh. 27 - Prob. 35PCh. 27 - Prob. 36PCh. 27 - Prob. 37PCh. 27 - Prob. 38PCh. 27 - Prob. 39PCh. 27 - Prob. 40PCh. 27 - Prob. 41PCh. 27 - Prob. 42PCh. 27 - Prob. 43PCh. 27 - Prob. 44PCh. 27 - Prob. 45PCh. 27 - Prob. 46PCh. 27 - Prob. 47PCh. 27 - Prob. 48PCh. 27 - Prob. 49PCh. 27 - Prob. 50PCh. 27 - Prob. 51PCh. 27 - Prob. 52PCh. 27 - Prob. 53P
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Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biochemistry and related others by exploring similar questions and additional content below.Similar questions
- Epinephrine binding allows the "fight-or-flight" response. Think about how this would differ from insulin binding. In the muscle, what would you expect for pathways use of glucose 6-P? Select all that apply. a. Glycogen breakdown is activated to produce glucose 6 - phosphate b. Glucose 6-phosphatase is activated to allow glucose release from muscle cell c. Pyruvate kinase is stabilized in the T-state d. Glycogen synthesis is activated e. PRK is stabilized in the R-state, glycolysis is activatedarrow_forward29. Synthesis signal. How does insulin stimulate glycogen synthesis?arrow_forwardInsulin - For the hormone Insulin (in humans) what is; 1. the origin (gland that secretes the hormone) 2. action of the hormone 3. source of control (how is the hormone regulatedarrow_forward
- True or false. Are the 3 hormones involved in restoring homeostasis after an intense exercise epinephrine, cortisol and insulin? Explain.arrow_forwardPancreas. Note the exocrine pancreas and the Islets of Langerhan’s also known as the Pancreatic Islets. Which part of the pancreas is endocrine? Label the two cells of the Pancreatic Islet: the alpha and beta cells (you cannot tell them apart unless especially stained). Also, include the delta cells. They produce somatostatin. What does this hormone do?arrow_forwardNeed answer of it. Name in detail each hypothalamus hormone and their function.arrow_forward
- Choose the correct. Insulin inhibits this catabolic process:A. Conversion of protein to aminoacidsB. Conversion of aminoacids to proteinsC. Conversion of glucose to fatty acidsD. GlycogenesisE. Conversion of fatty acids to triglyceridesarrow_forwardInsulin increasesa. the uptake of glucose by its target tissues.b. the breakdown of protein.c. the breakdown of lipids.d. glycogen breakdown in the liver.e. All of these are correct.arrow_forwardConsidering the endocrine function of adipose tissue, why is it so hard for people to maintain weight loss? I was thinking from the adipose tissue there is: adiponectin → hormone synthesized by adipose cells → reduce cellular insulin resistance + protects blood vessels from inflammation + atherosclerosis. However, the levels of the hormone are lower in people who are obese. But I'm not sure if that is correct or not.arrow_forward
- Explain why insulin is required for adipocytes to synthesize triacylglycerols from fatty acids.arrow_forwardA tiger is stalking you. Explain how your metabolism respond to help you escape. Include: a) which metabolic pathways are activated/deactivated and describe the pathways in the context of individual tissues. b) the hormones that activate those pathways and how (chemically) the hormones regulate each pathway c) an explanation for why those pathways need to be active in order for you to escapearrow_forwardPeople with non-insulin-dependent diabetes mellitus are generally not ketosis prone. This is thought to be a result of: the lack of increase in glucagons in the se individuals the presence of insulin in the individuals their obesity the fact that, their blood glucose levels do not tend to rise significantly Metabolic actions of insulin include all of the following except: increased glycogenesis decreased gluconeogenesis increased basal metabolic rate increased skeletal muscle amino acid up-take all of the above are metabolic actions of insulinarrow_forward
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