Concept explainers
To determine: The physiological justification of the treatment for atrial fibrillation and the reason for the harmful effects of rapid atrial depolarization.
Introduction: In the new treatment of atrial fibrillation, high voltage electric impulse is administrated to control the rapid rate at senatorial (SA) node and destroy the autorhythmic cells of the atrioventricular (AV) node. After that, the ventricular pacemaker is implanted in the patient.
To determine: The reason for the destruction of the atrio-ventricular (AV) node in the procedure to treat atrial fibrillation.
Introduction: Cardiac muscles fiber of heart joins with each other to form a network. This network of muscle fiber is called myocardium. The muscles of the heart are involuntary and do not get tired. Therefore, the action potential of myocardial muscle is different from other muscle fibers.
To determine: The reason for the implantation of a pacemaker in the procedure to treat atrial fibrillation.
Introduction: Cardiac muscles fiber of the heart joins with each other to form a network. This network of muscle fiber is called myocardium. All the myocardial muscles generate action potential simultaneously to initiate electrical conduction the heart.
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Human Physiology: An Integrated Approach (8th Edition)
- 1)An increase in total peripheral resistance, all else remaining the same, will cause an increase in mean arterial pressure.True/False 2) We care about mean arterial pressure specifically because we care about having an appropriate rate of blood flow through capillaries. True/False 3) A person's L-type channels are mutated so that they do not inactivate. How would this affect the time duration of a cardiac action potential? Group of answer choices a)Increase b)No change c)Decrease 4) A person' L-type channels are mutated so that they do not inactivate. How would this affect the time duration of a cardiac muscle cell contraction? Group of answer choices a)Increase b)No change c)Decreasearrow_forwardDraw these five graphs on the same set of axes: atrial autorhythmic cell action potential atrial cardiac contractile cell action potential ventricular autorhythmic cell action potential ventricular cardiac contractile cell action potential ventricular myocardial muscle tensionarrow_forwardWhat are the difference between contractile cells of the heart and electrical cells responsible for generation and conduction of impulses. Why is the cardiac action potential propagated more slowly in an AV node cell than in an atrial or ventricular myocyte? How are the channels and ions that are responsible for depolarization and repolarization work? How can be AV conduction affected by hypokalemia?arrow_forward
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