Testerone is a steriod (lipid) hormone. Which of the following would be the most likely receptor for this hormone? Select one: a. A transmembrane receptor in the cell membrane since it cannot diffuse across the cell membrane b. An intracellular receptor because it can diffuse across the cell membrane

In their mechanism of action, a difference between steroid and nonsteroid hormones is that __________. nonsteroid hormones bind to DNA steroid hormones enter the nucleus of the target cell, and nonsteroid hormones do not nonsteroid hormones cross the plasma membrane more readily than do steroid hormones steroid hormones use a signal transduction pathway

Why is the fact that a monomeric hormone binds simultaneously to two identical receptor molecules, thus promoting the formation of a dimer of the receptor, considered remarkable?

Which of the following is not a true statement? A.) steroid and thyroid hormone signaling would be impaired if the plasma level of carrier proteins is not optimal. B.) Steroid and thyroid hormone-receptor complexes form dimers when they bind to the hormone-response elements on DNA C.) Epinephrine uses the adenylate cyclase and the phospholipase C second messenger systems D.) signaling by a peptide hormone would be impaired if a second messenger system is not functional in the target cell E.) signaling by a steroid hormone would be hindered if the adenylated cyclase second messenger system is not functional in the target cell.

Can you please describe the role of G proteins in ligand-activated cellular signal transduction.

A Moving to the next question prevents changes to this answer. Question 1 What is unusual about the receptor for steroid hormones? A. The receptor is located in the cytoplasm but can enter the nucleus once bound to ligand. B. The receptor is embedded in the plasma membrane C. The receptor is located in the cytoplasm but can never enter the nucleus. D. The receptor is located in the lysosome. A Moving to the next question prevents changes to this answer.

Why are the catecholamines listed for a variety of receptor pathways?

Steroid hormones are required by the body at puberty and into adolescence to regulate growth and cell division at more rapid pace than in later life.  This regulation occurs via their interaction with cellular receptors and the signaling cascades/pathways that follow.  Describe for me the difference between the two major classes of steroids, anabolic and catabolic steroids.  What might you expect the result of signaling cascades to be in cells receiving either anabolic or catabolic “signals”? (B) At some point in late adolescence, steroid production decreases by almost 100 fold, as we transition into “adulthood”.  Why might we wish to stop these signals from constantly being in our blood stream, (like, Say, between 17-24 years of age)?  What result might these steroids have on cancer cells where abhorrent signaling is already causing an increased rate of cell division/growth? Could steroid use result in Cancer?

A biochemist is designing a new drug that will mimic the shape and function of a particular intercellular signaling molecule. Therefore she must produce a molecule that will bind to the receptor protein and... A. produce a cellular response by altering the Na+/K+ pump located near the receptor site. B. induce a change in the receptor molecule shape, which will ultimately prevent a cellular response. C.  induce a change in the receptor molecule shape, which will ultimately produce a cellular response. D.  induce a change in the phospholipid bilayer that causes a polar reversal allowing the molecules to be endocytose (engulfed).

Which of the following best describes the type of receptor a beta-antagonist drugs acts on a.Ligand-regulated transmembrane enzymes b.Ion channel c.G protein-coupled receptor d.Intracellular lipid-soluble signaling