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Why are mutations in the INK4 locus so dangerous?
Step by step
Solved in 2 steps
- Why are the correct primer sequences essential for successful amplification?Why is the p53 R273W mutation preventing p53 from binding to DNA?As described in Table, what is the difference between a rapidstop and a slow-stop mutant? What are different roles of the proteins that are defective in rapid-stop and slow-stop mutants?