What happens if there is too much p16-ink4a in a juvenile and elderly human? what happens if a juvenile produces a mutation form that lacks activity?
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What happens if there is too much p16-ink4a in a juvenile and elderly human?
what happens if a juvenile produces a mutation form that lacks activity?
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- Two possible point mutations are the substitution of lysine for leucine or the substitution of serine for threonine. Which is likely to be more serious and why?Why would a mutation in a somatic cell of a multicellular organism escape detection?The genetic alteration responsible for sickle-cell anemia in humans involves: a transition mutation from A to G, substituting glutamic acid for valine in a-globin a transversion mutation from T to A, substituting valine for glutamic acid in b-globin a transition mutation from T to C, substituting valine for glutamic acid in b-globin a transversion mutation from G to C, substituting glutamic acid for valine in a-globin a frameshift mutation of one ATC codon, removing glutamic acid from b-globin
- The most prominent mutation of ERBB2 in breast cancer is L755S. What kind of mutation is this? Is this a driver or passenger mutation? How to do you know?Why are mutations in the INK4 locus so dangerous?What is the probability that the palindromic symmetry of the trp repressor target DNA sequence is merely accidental?
- PTC is not a biological compound, but a chemical which became known after research done by Arthur J. Fox. Why is the TAS2R38 gene also called the PTC gene?Among the different types of amino acid substitution (same sense, missense, or nonsense mutations), which is the most deleterious one?What is the difference between a missense mutation and a nonsense mutation?