Explain how the technique of replica plating supports the randommutation theory but conflicts with the physiological adaptationhypothesis.

why does a clone of cells produceonly one type of G-6-PD enzyme? What would you expect to happen if a clone was derived from an early embryonic cell? Whydoes the initial sample of tissue produce both forms of G-6-PD?

6. Consider a scenario in which allele A2 is recessive and it decreases fitness (i.e. it is deleterious). What is the ratio of the frequency of the deleterious recessive homozygote in an inbred versus a non-inbred (i.e. random mating) population if the frequency of the deleterious allele is q = 104 and f=0.01? 7. This question will not be marked but I strongly encourage to you to do it nevertheless. It uses a simulation of genetic drift that may help your conceptual understanding of the process. Download and install Populus (http://cbs.umn.edu/populus/download-populus). See here if you have problems installing or running it (https://cbs.umn.edu/populus/contact). Press the Model button and select Mendelian Genetics, then Genetic Drift. Select the Monte Carlo tab. This is a simulation method used to implement a Fisher-Wright model of drift. (You can read the model background in the Populus help document if you're interested.) The initial dialog has values for run time (i.e. # of…

Based on Pruisner (2013), match each of the following terms with its appropriate definition or description.                                                                         PRNP gene                                                                        CPEB (cytoplasmic polyadenylation element binding) protein                                                                        NFT (neurofibrillary tangle)                                                                        A673T-mutated AAP                                                                        MAPT gene A. a "nonpathogenic prion" B. encodes PrPC (the normal cellular form of prion protein) C. encodes tau protein;  mutations in this gene underlie many familial FTDs (frontotemporal dementias) D. tau-rich fibril associated with many neurodegenerative diseases E. a missense mutation in β-secretase which appears to protect against late-onset…

Differentiate the lock-and-key model from the induced fit hypothesis

When performing QPCR to study differential gene expression, what is the role of SYBR green? What is the most correct answer? SYBRgreen intercalates proportionally and efficiently with double stranded DNA and can therefore be used to quantify it   SYBRgreen intercalates proportionally and efficiently with nucleic acids and can therefore be used to quantify them.   SYBRgreen intercalates more efficiently and proportionately with single stranded DNA and can therefore be used to quantify cDNA (a direct product of mRNA).   SYBRgreen intercalates proportionally efficiently with messenger RNA and can therefore be used to quantify it.

If A-P width is the most important regulator of digit number, which basic cellular mechanism would you expect to be correlated with digit number?

I need to know the answer to how these are sorted. Somehow, my original answer was incorrect (mastering says 2 of them were out of place) - but in the reading from the book and lecture notes, I truly have no clue where I'm going wrong here.   Please help!  The one image is just the original problem, and the second was my attempt (where 2 were incorrect). Thank you!

Glomerular filtration can be increased by which of the following? A. Increasing blood flow via afferent arteriole B. Decreasing blood flow via afferent arteriole C. Decreasing arterial blood pressure D. Increasing blood flow via efferent arteriole The answer is (A) Increasing blood flow via afferent arteriole. Please explain why it is not the other options (B,C,D)

Give typing answer with explanation and conclusion  What is the transcription product of the DNA templa GCTAGCGATGAC-5'? OA) CAGTAGCGATCG OB) CGAUCGCUACUG OC) CGATCGCTACUG OD) CGUTCGCUTCUG