BIOLOGY:CONCEPTS+APPL.(LOOSELEAF)
BIOLOGY:CONCEPTS+APPL.(LOOSELEAF)
10th Edition
ISBN: 9781305967359
Author: STARR
Publisher: CENGAGE L
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Chapter 7, Problem 1CT
Summary Introduction

To determine:

The reason that cyanide poisoning starts with the same symptom as altitude sickness.

Introduction:

The people who live at higher altitudes face some medical problems. They suffer from weakness, dizziness and so on. They even suffer from breath shortness. However, people suffering from cyanide poising also show the same symptoms. It has been discovered that the cyanide has the ability to bind to a molecule called cytochrome c oxidase. The binding of these two units reduces the concentration of oxygen present in mitochondria of the cell. As a result, this type of bonding of cytochrome c oxidase and cyanide inhibits the transfer of electrons inside mitochondria.

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The higher the altitude, the lower the oxygen level in air. Climbers of very tall mountains risk altitude sickness, which is an illness characterized by shortness of breath, weakness, dizziness, and confusion.  The early symptoms of cyanide poisoning are the same as those for altitude sickness.  Cyanide binds tightly to cytochrome c oxidase, the protein that reduces oxygen molecules in the final step of mitochondrial electron transfer chains.  Cytochrome c oxidase with bound cyanide can no longer transfer electrons.  Explain why cyanide poisoning starts with the same symptoms as altitude sickness.
The higher the altitude the lower the oxygen level in air. Climbers of very tall mountains risk altitude sickness which is characterized by shortness of breath, weakness, dizziness, and confusion. The early symptoms of cyanide poisoning are the same as those for altitude sickness.    Cyanide binds tightly to cytochrome c oxidase, a protein complex that is the last component of the mitochondrial electron transfer chain. Cytochrome c oxidase with bound cyanide can no longer transfer electrons.  Explain why cyanide poisoning starts with the same symptoms as altitude sickness.
Mutations that prevent Bax and Bak from interacting with the outer mitochondrial membrane would prevent the release of cytochrome c and inhibit intrinsic apoptosis. prevent the release of cytochrome cand promote assembly of the apoptosome. trigger the release of cytochrome c and promote intrinsic apoptosis. trigger the release of cytochrome c and promote the assembly of the apoptosome. trigger the release of cytochrome c and promote the cleavage of procaspase-8.
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