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Why is it beneficial for CO2 chemoreceptor sensitivity to be increased when a woman is pregnant?
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- Is the chemical signal that causes the cessation of breath holding a decrease or an increase in CO2 in the blood?Which option best describes the following figure? Y (fraction saturation) 1.0 0.8 0.6 0.4 0.2 0.0 0 20 Oxygen Binding plot 40 60 p02 (torr) 80 100 A) Curve x = adult haemoglobin, curve y = fetal haemoglobin B) Curve x = myoglobin, curve y = haemoglobin C) Curve x = fetal haemoglobin, curve y = adult haemoglobin D) Curve x = fetal haemoglobin, curve y = myoglobin E) Curve x = adult haemoglobin, curve y - myoglobinGiven the difficulty in conducting a manipulative experiment to identify the effects of elevated CO2 across the globe, how might we be able to validate the mathematical models that have been created?
- Below is the equation that represents O2 binding to hemoglobin. Hb+4O2⇌Hb(O2)According to Le Chatelier’s principle, which form of the hemoglobin (Hb or Hb(O2)4) is predominant in the lungs where the partial pressure of oxygen is high? Explain. I need help answering thisCan you please answer this question please. Does the change in HR affect CO? Please explain how.Below is a fractional saturation curve for O₂ binding to adult hemoglobin. Assume that curve Y represents a condition at pH 7.4 and with a normal physiological level of 2,3-BPG. Curve X represents a condition that Fractional saturation (0) 0.2 0.0 0 Tissue PO, (kPa) 12 Lungs
- A newborn exhibits signs of respiratory distress due to the lack of surfactant. She has signs of low oxygen and high carbon dioxide. Explain why the lack of surfactant would result in respiratory distress.How does the movement of CO2 from fetal blood into maternal bloodincrease the movement of O2 from maternal blood into fetal blood?(Hint: Consider the shift of the oxygen-hemoglobin dissociation curve.)You have both peripheral chemoreceptors (aortic arch and carotid bodies) and central chemoreceptors (medulla). You were informed earlier that the central chemoreceptors are the primary sensors for blood CO2. Both sets of chemoreceptors detect the concentration of H+ in blood or cerebral spinal fluid (CSF). Why do the central chemoreceptors detect blood PCO2 levels while the peripheral chemoreceptors are only sensitive to total blood H+ concentration?