Which component of cell division machinery is frequently targeted by anti-cancer drugs? Can you explain the common side-effects of chemotherapy (e.g. hair loss, mucositis) based on this information?
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Which component of cell division machinery is frequently targeted by anti-cancer drugs? Can you explain the common side-effects of chemotherapy (e.g. hair loss, mucositis) based on this information?
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- name any two methods that can possibly cure cancer .Name the six fundamental properties of malignant tumours. Which of these properties are amenable to study in a cell culture model of cancer and why?The best strategy for treating a specific type of human tumor can depend on identifying the type of cell that became cancerous to give rise to the tumor. For some tumors that have colonized a distant location (metastasized), identifying the parental cell type can be difficult. Because the type of IF protein expressed is cell-type-specific, using monoclonal antibodies that react with only one type of IF protein can help in this identification. What IF proteins would you produce monoclonal antibodies against to identify (a) a sarcoma of muscle cell origin, (b) an epithelial cell carcinoma, and (c) an astrocytoma (glial cell tumor)?
- Which of the following drugs would directly target the ability of cancer cells to evade cell growth suppressors? (evading the growth suppressor hallmark) Anti-inflammatory drugs VEGF signaling inhibitors Cyclin-dependent kinase inhibitors Telomerase inhibitorsJurkat cells are an immortalized line of human T-lymphocytes cells that are used to study acute T cell leukemia. K562 cells were the first human immortalized myelogenous leukemia cell line to be established. Resting= control, Etoposide= drug. Below is shown the results of floweytometry of untreated and treated cells.The growth fraction of the cancer types A 8, C, and D are 23%, 45%, 56%, and 78%, respectively. Which type of cancer is more suceptible to the chemotherapy?
- Tissues and differentiation a)Explain what is meant by termination and differentiation ).b) Explain the difference between an oncogenic and a tumour suppressor gene and describe how they are involved in the onset of cancerDiscuss why certain cell types are less likely and others are more likely to develop cancer than others.Which of the following is true of tumor suppressor genes? Group of answer choices a) If this gene is overactive, it becomes an oncogene b) If one of the alleles is mutated, there is usually little effect. Two inactivating mutations are usually required for loss of function (recessive mutation). c) If one copy is lost, the gene no longer functions (dominant mutation) d) Tumor suppressors genes usually cause mitosis or cell growth e) Tumor suppressor genes decrease apoptosis
- Describe the general process of cell signalling pathways: what events take place for a signal to cause cellular changes? Provide examples and how perturbation of these events can result in “cancer pathways”. In addition, describe in detail a typical cancer pathway and its strategy to activate gene expression. What is the origin of many cancer pathways, i.e., during which stage of an organism’s live process(es) are they physiologically activated? Why is this important for cancer development?Why is it important to model cancer through the generation of induced pluripotent stem cells ? Explain in detail the main findings.Explain what is fundamental aberrations in all cancer cells ?