The symptoms of Cushing’s disease include abdominal obesity, hypertension, glucose intolerance (steroid diabetes), hirsutism, osteoporosis, polyuria, and polydipsia. Describe the consequences of cortisol excess that would produce each of these symptoms. Which tests can be performed to determine if a patient has primary or secondary Cushing’s disease? What would the results of these tests be for each type of disease?

An unresponsive client who has diabetes is brought to the emergency department with rapid, deep respirations. Additional findings include: blood glucose 24.9 mmol/L, arterial pH 7.2 and urinalysis showing presence of ketones and glucose. Which of the following statements best describes the underlying cause of this patient’s presentation? Question 64 options: a)  Relative insulin deficiency, causing hyperglycemia, oxidative stress, renal dysfunction and acidosis b)  Nocturnal elevation of growth hormone resulting in hyperglycemia in the morning c)  Absolute insulin deficiency, increased counter-regulatory hormone, lipolysis and free fatty acid release d)  Hypoglycemia causes release of glucagon, resulting in glycogenolysis and hyperglycemia

Jessica Sagat, 21 years old, is a type 1 diabetic client. She has been ill for the past several days and unable to control her blood sugar. She has developed elevated blood sugar, nausea, vomiting, rapid and deep respirations, and a fruity odor to her breath. An arterial blood gas (ABG) is taken and the results are: pH 7.30, PaCO2 43 mm Hg, Bicarb (HCO3) 19 mEq/L, and PaO2 93 mmHg. What type of acid–base imbalance is this client experiencing? What components of the acid–base balance system are involved in this imbalance? What are the normal ABG values?

describe differences between Cushing syndrome and Cushing disease.  Keep in mind that the most important aspect here is understanding the organs and hormones that are involved.

What is the genetics: prevalence, hereditary pattern of inherited metabolic diseases of Congential Adrenal Hyperplasia.

What two imbalances is a patient at risk for when they have an exacerbation of IBD?

after some research, i concluded the final diagnosis is due to rickets, for differential diagnosis, i want to speak about; primary hyperparathyroidism, Paget's disease, osteomalacia, and rickets. what I'm struggling with is i want to explain why the underlying biochemistry and cellular pathology of the diseases. and explain WHY there is a difference between these diseases and how the analytes and potential explains it

In a patient with suspected primary hyperthyroidism associated with Graves' disease, one would expect the following laboratory serum results: T4 _________, TSH _________. 1) Increased, increased 2) Decreased, normal 3)  Increased, normal 4)  Increased, decreased 5)  Normal, increased no references, just homework.

From the following list, choose the THREE things that would occur as a result of a patient becoming hypocalcemic.   increased urinary excretion of calcium increase in PTH reduction in osteoclast activity increased osteoblast activity increase in calcitriol reduction in osteoblast activity increased deposition of hyaline cartilage

Describe the homeostatic control of plasma calcium levels by answering the following two questions: What role does calcitonin play in the maintenance of serum calcium? Give reference to the gland involved in secretion, the bone cells stimulated by calcitonin and the effects of calcitonin.  Describe the role parathyroid hormone (PTH) has in the maintenance of serum calcium, giving reference to the gland involved, the particular bone cells stimulated and the major effects of PTH.