I've been confused about diabetes lately. I know that it's said that the pancreas produces insulin, but someone said that actually the pancreas produces beta and alpha cells that signals for the production of insulin or release of glucagon from another organ/gland. Which is it?

Which of the following is a consequence of insulin signaling? (select all that apply)     Cell growth/division genes upregulated     Increased cellular glucose uptake through GLUT4 transporter     glycogen degradation     Glucose synthesis

The hormones insulin and glucagon play an important role in the regulation of plasma glucose. b) People with Type II diabetes mellitus can manage their condition (maintain adequate glycaemic control) through use of oral hypoglycaemic drugs. Provide a rationale for how two hypoglycaemic drugs, with different mechanisms of action, can both work to lower blood glucose levels in Type II diabetes.

For our patient John with diabetes type II, he can make insulin but his body cells can be resistant to this insulin. Note: activated insulin receptors will activate glucose receptors. John takes a drug called metformin, in conjunction with regulating his diet and exercising. This drug has two main effects. First, it helps reduce hepatic (liver) production of glucose. Second, it helps cells become more sensitive to insulin. Explain how these two effects of metformin can help John.

Which of the following sequences is correct? Corticotropin-releasing hormone (CRH), adrenocorticotropic-releasing hormone (ACTH), adrenal cortex. Corticotropin-releasing hormone (CRH), adrenocorticotropic-releasing hormone (ACTH), adrenal medulla. Adrenocorticotropic-releasing hormone (ACTH), corticotropin-releasing hormone (CRH), adrenal medulla. Adrenocorticotropic-releasing hormone (ACTH), corticotropin-releasing hormone (CRH), adrenal cortex.

What are some examples of parasitism?

1.20-year-old woman with diabetes mellitus was hospitalized with confused consciousness,fever,nausea,vomiting and fruity smelling breath. A urine sample was strongly positive for ketone bodies.Why would an insulin injection decrease her ketone body production? For the answer: a) indicatethe causes of diabetes mellitus:describe the scheme of signal transduction by insulin. b)compare the blood glucose levels in healthy patients and in patients with diabetes mellitus; c)explain why ketone bodies synthesis is activated in this patient: describe the schemes of ketone bodies synthesis and oxidation.

Considering the endocrine function of adipose tissue, why is it so hard for people to maintain weight loss? I was thinking from the adipose tissue there is: adiponectin → hormone synthesized by adipose cells → reduce cellular insulin resistance + protects blood vessels from inflammation + atherosclerosis. However, the levels of the hormone are lower in people who are obese. But I'm not sure if that is correct or not.

Under what conditions would insulin be released? Is this a hormonal or humoral factor?

Alpha cells in the pancreas secrete in response to blood sugar levels, which is defined as and likely happens after we are Beta cells in the pancreas secrete in response to blood sugar levels, which is defined as and likely happens after we are Hyperglycemia Hypoglycemia Eating Insulin Exercising/Fasting Low Glucagon High