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Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN: 9780134580999
Author: Elaine N. Marieb, Katja N. Hoehn
Publisher: PEARSON
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
Transcribed Image Text:It is able to bind with peptidase enzymes preventing bacterial call wall
synthesis.
8. Explain how antibiotics and/or antibiotic resistance are examples of the statement
"structure determines function."
Watch th video posted on
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Antibiotics are a class of drugs that are used to treat bacterial infections. They work by either killing bacteria or preventing their growth and replication
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- Which of the following does NOT describe a mechanism of antibiotic resistance among bacteria? enzymes to hydrolyze or chemically alter the antibiotics mutations in target molecules so that antibiotics can no longer bind efflux pumps cell wall diffusion barrier, e.g. Gram-negative outer membrane, Acid Fast cell wall of Mycobacterium O increased synthesis of porins to increase antibiotic diffusion across membranesarrow_forward"On the rapidity of antibiotic resistance evolution facilitated by a concentration gradient" The rapid emergence of bacterial strains resistant to multiple antibiotics is posing a growing public health risk. The mechanisms underlying the rapid evolution of drug resistance are, however, poorly understood. The heterogeneity of the environments in which bacteria encounter antibiotic drugs could play an important role. E.g., in the highly compartmentalized human body, drug levels can vary substantially between different organs and tissues. It has been proposed that this could facilitate the selection of resistance mutants, and recent experiments support this. To study the role of spatial heterogeneity in the evolution of drug resistance, we present a quantitative model describing an environment subdivided into relatively isolated compartments with various antibiotic concentrations, in which bacteria evolve under the stochastic processes of proliferation, migration, mutation and death.…arrow_forwardHello just wondering, what do we look for in bacteria we don't have in regards to this question?arrow_forward
- 12. Penicillin only works on actively dividing bacterial cells because: a. it inhibits peptidoglycan (PG) synthesis b. it inhibits LPS synthesis c. it is no longer is effective against RNA and translation d. a & b e. a & carrow_forwardusing the figure, provide a size estimate fo (1) a virus, (2) a prokaryotic cell, and (3) a eukaryotic cell. be sure to include units!arrow_forwardSome antibiotics target peptidoglycan synthesis. What is amolecular growth target of an antibiotic that inhibitspeptidoglycan synthesis?arrow_forward
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