eve stripe 2 reporterABHunchbackCD6999KruppelEFDIn the early Drosophila embryo, the even-skipped (eve) gene is expressed in a stripedpattern. eve stripe #2 expression is controlled by a regulatory DNA sequence thatincludes binding sites for the regulatory proteins Hunchback and Kruppel. The boxedfigure at left above shows the expression patterns, in the early embryo, of a reportergene joined to the eve stripe 2 regulatory sequence, and of the Hunchback andKruppel proteins. Which of the figures at right best predicts the expression patternof the eve stripe2 reporter (blue) in a mutant embryo lacking the Kruppel protein?The dashed lines in each outline the eve stripe 2 position in a wild-type embryo. ○ A) No change from wild typeB) No reporter gene expressionC) Reporter expression is expanded, to the anterior end of the embryoD) Reporter expression is expanded, in the posterior direction.○ E) Reporter expression is shifted, to the anterior end of the embryo○ F) Reporter expression is shifted, in the posterior direction

Option b: No reporter gene expression. This option is correct or incorrect because the krupple…

Answered: eve stripe 2 reporter A B Hunchback C D…

Human Heredity: Principles and Issues (MindTap Course List)

11th Edition

ISBN:9781305251052

Author:Michael Cummings

Publisher:Michael Cummings

Chapter15: Genomes And Genomics

Section: Chapter Questions

Problem 13QP

See similar textbooks

Similar questions

Recently, scientists constructed a transgene that expresses a mutant form of Drosophila histone H3 inwhich lysine 27 in the histone tail was changed to methionine (H3K27M). Expression of the H3K27Mtransgene results in aberrant development of fruit fliesbecause of inappropriate expression of many differentgenes. Explain this finding.
What would be the most likely effect of inhibiting the translation of hunchback mRNA throughout a Drosophila embryo?
Telomere position effect (TPE) alters the gene expression in yeast cells? why?
Recall that the nuclear membrane disintegrates late in prophase of mitosis in most eukarvotic cells. Once the membrane is reformed in telophase in a daughter cell, several components of gene expression mignt therefore be "caught" out in the cytoplasm when they would otherwise onlv ever be found inside the nucleus. Consider where the following components of gene expression are made and where they runction. Which component is normally never found in the cytoplasm outside the nucleus? O A. Spliced intron • B. RNA polymerase O C. Histones • D. DNA polymerase
You have been analyzing wing development mutants in Drosophila and have collected the genetic/Western data below. Your epistasis analysis (not shown), suggested your current model that DW-2 is upstream of WL-1. What is a plausible mechanism for how DW-2 regulates WL-1? mutant name wingless-1 doublewing-2 Prated with Ants-Wingless MW standards || | | mutanttype ODW-2 inhibits transcription of WL-1 O DW-2 polyubiquitinates WL-1 null null phenotype Model Ac O DW-2 phosphorylates WL-1 and prevents it from entering the nucleus O DW-2 cleaves WL-1 proteolytically Wings Mode of regulation DW-2 Wings Wings
Based on Figure 14-14 and the features of ultraconservedelements, what would you predict you’d observe if youinjected a reporter-gene construct of the rat ortholog ofthe ISL1 ultraconserved element into fertilized mouseoocytes and examined reporter gene expression in thedeveloping embryo?
Polarity of oocyte of DrosophilaWhat are the cells surrounding the oocyte? Are they germ-line or somatic in origin?
Polarity of oocyte of DrosophilaWhat is the difference between the anterior and posterior determinant for the oocyte?
The extracellular protein factor Decapentaplegic(Dpp) is critical for proper wing development in Drosoph-ila (Figure Q21–3A). It is normally expressed in a narrowstripe in the middle of the wing, along the anterior–pos-terior boundary. Flies that are defective for Dpp formstunted “wings” (Figure Q21–3B). If an additional copyof the gene is placed under control of a promoter that isactive in the anterior part of the wing, or in the posterior part of the wing, a large mass of wing tissue composed ofnormal-looking cells is produced at the site of Dpp expres-sion (Figure Q21–3C and D). Does Dpp stimulate cell divi-sion, cell growth, or both? How can you tell?
In mammalian females, the majority of one X chromosome's genes are shut down in a process called X chromosome condensed and afterwards called a The inactive X is Because the shutdown is random in the embryonic cells formed by the time inactivation occurs, all mature mammalian females are a for the expression of X chromosome genes. The inactivation of one X in the female is thought to . The shutdown of the X chromosome is accomplished by the balance gene expression between males and females, a theory called gene. is the master gene for male sex determination in mammals and is found on the human Y-chromosome.
Which of the following proteins is a combinatorial transcriptional regulator in Drosophila that affects the differentiation of multiple cell types? O the glucocorticoid receptor O MyoD O Ey O Muts O maintenance methyltransferase
In the following study, the investigators wanted to determine the role of cyclin B in controlling the cell cycle. Earlier researchers had found that extracts made from frog eggs (Xenopus) contained all the necessary proteins and machinery required for DNA replication. This included proteins that regulated the mitosis promoting factor (MPF). At the time of this study, cyclin B was show to affect MPF activity and the research group wanted to test using Xenopus egg extract in an assay. In Figure 1 (a) MPF activity was tested for its ability to phosphorylate Histone (H1) in sperm chromatin over a certain period of time. Additionally, the cyclin B concentration in the extract was measured. In Figure1c, Only cyclin B MRNA was added back to the RNase-treated extract and the assay was continued. What would Figure 1c seem to suggest to the authors?

SEE MORE QUESTIONS

Recommended textbooks for you

Human Heredity: Principles and Issues (MindTap Co…

Biology

ISBN:

9781305251052

Author:

Michael Cummings

Publisher:

Cengage Learning