What is the net reaction of the citric acid cycle? What happens to each product?OXIDATIVE PHOSPHORYLATION:3A) Thoroughly explain the biological significance of NADH/H* and FADH₂ and their roles inoxidative phosphorylation.3B) Describe and diagram the general pathway of the electrons from NADH/H+ and FADH2 to 02during the electron transport chain.3C) What is "chemiosmotic coupling", and what is its relationship to ATP synthesis?3D) During oxidative phosphorylation, what is oxidized and what is phosphorylated?

All life forms require energy. We obtain this energy from the food we eat. It is the highly reduced…

Answered: OXIDATIVE PHOSPHORYLATION: 3A)…

Biochemistry

9th Edition

ISBN:9781319114671

Author:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Publisher:Lubert Stryer, Jeremy M. Berg, John L. Tymoczko, Gregory J. Gatto Jr.

Chapter1: Biochemistry: An Evolving Science

Section: Chapter Questions

Problem 1P

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DCCD (diocyclohexylcarbodiimide) inhibits oxidative phosphorylation when the substrate is mitochondrial NADH. DCCD is a drug that binds to ATP synthase and blocks proton transport through the ion channel. a) Explain what the consequences of DCCD on cellular energy production are. b) Suggest at least one other cellular effect of DCCD and explain this effect.
The functioning of a particular pathway often depends not only on control enzymes in that pathway but alsoon control enzymes of other pathways. What happens in the following pathways under the indicated conditions? Suggest what otherpathway or pathways might be influenced.(a) High ATP or NADH concentration and the citric acid cycle.(b) High ATP concentration and glycolysis.(c) High NADPH concentration and the pentose phosphate pathway.(d) High fructose-2,6-bisphosphate concentration and gluconeo genesis.
Under standard conditions, is the oxidation of ubiquinol (Coenzyme Q) by O2 sufficiently exergonic to drive the synthesis of ATP? If yes, how many ATP can be synthesized assuming 100% efficiency?
how many net ATP molecules does the citric acid cycle generate in total? Consider both ATP generated directly by the citric acid cycle and ATP generated downstream in oxidative phosphorylation using other molecuies generated by the citric acid cycle For your answer, consider ATP and GTP to be equivalent A) 32 B) 20 C) 2 D) 10 E) 1
Describe how a) Mean Arterial Pressure, b) Maximal Oxygen Consumption, and c) Blood Flow are determined using its formula. In addition, you should explain what those factors are including cardiac output, stroke volume, heart rate, resistance, a-vO2 difference, etc. Describe how many ATPs can be generated by complete cycles of beta oxidation of free fatty acid with 20 carbons. You should indicate how many cycles of beta oxidation and Krebs cycle, and total number of products as well
this question is about 16 carbon fatty acid A) what are the end procuts of the eight cycles of beta oxidation of a 16 carbon fatty acid? B) assuming 2.5 ATP molecules are produced from one NADH, how many molecules of ATP are produced from the NADH produced during beta oxidation of a 16-carbon fatty acid
the reaction catalyzed by glyceradehyde 3-phosphate dehydrigenase is based on NAD+ and a active site cysteine. Also another phosphate group is added. what is the reason for that? a) because one ATP is consumed b) an inorganic phosphate is activated for ATP synthesis C) NADH can be recycld and than converted back to NAD+ for glycolysis d) because one ATP is generated
match the cofactor with its function in the citric acid cycle by entering the number corresponding with the function. A given function may be used more than one time or not at all. Use from the following list of functions to fill in the table below: carries O2 carries small carbon-containing molecules carries e- carries small nitrogen-containing molecules Cofactor Function NAD+/NADH FAD/FADH2 CoA thiamine biotin
You are limited to the carbon in the following molecules: One xylulose 5-phosphate, One glyceraldehyde 3-phosphate, One sedoheptulose 7-phosphate, 1 Oxaloacetate, and 3 carbon dioxide A) Disregard regulation completely regarding pathway activity, using only the enzymes of glycolysis, pentose phosphate pathway, and citric acid cycle, what is the maximum ATP one can generate with these molecules? B) Disregard regulation completely regarding pathway activity, using only the enzymes of glycolysis, Calvin-Benson-Bassham cycle, and citric acid cycle, what is the maximum ATP one can generate with these molecules (in this instance only, you also are given 3 ATP and note that you do NOT need to regenerate substrates for rubisco once you get through this enzyme).
1) Using the equations for cellular respiration and photosynthesis, explain what makes one pathway overwhelmingly anabolic and the other overwhelmingly catabolic. 2) Write out the equation for cellular respiration in words. What molecule is reduced and which is oxidized? 3) What goes into, and what comes out of the four steps of cellular respiration (glycolysis, pyruvate oxidation, the citric acid cycle, electron transport chain)? 4) Differentiate between and describe the three types of phosphorylation as discussed in the lecture and additional narrated whiteboard work available on in your "video material" for the week. You should go into detail here. Go into EXTREME detail with regard to oxidative phosphorylation. That will absolutely be represented in a BIG way on your next exam.
Discuss the composition of the transition state for the formation of ATP by ATP Synthase. a) Where is the active site for this enzyme located? b) How are the amino acid side chains from the α and β subunits of ATP Synthase involved? c) Discuss the importance of Mg+2 in the mechanism of this enzyme.
Step 6 of the citric acid cycle is shown. Which statement best describes the role of FAD in this reaction? CH₂ lal CH₂ FAD FADH₂ CO₂ CO₂ succinate CH CH fumarate OA) FAD causes the oxidation of succinate to form fumarate. B) FAD causes succinate to undergo hydration to form fumarate. C) FAD causes the isomerication of succinate to form fumarate. D) FAD causes the reduction of succinate to form fumarate.

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