Researchers are designing and testing antisense drugs as therapies for a variety of diseases, including cancer, AIDS, diabetes, and muscular dystrophy. The drugs are also being tested to fight infection by deadly viruses such as Ebola. Antisense drugs consist of short RNA strands complementary in sequence to mRNAs that form during the progression of a disease. How do you think these drugs work?

Researchers are designing and testing antisense drugs as therapies for a variety of diseases, including cancer, AIDS, diabetes, and muscular dystrophy. The drugs are also being tested to fight infection by deadly viruses such as Ebola. Antisense drugs consist of short RNA strands complementary in sequence to mRNAs that form during the progression of a disease. How do you think these drugs work?

Human Heredity: Principles and Issues (MindTap Course List)

11th Edition

ISBN:9781305251052

Author:Michael Cummings

Publisher:Michael Cummings

Chapter11: Genome Alterations: Mutation And Epigenetics

Section: Chapter Questions

Problem 6QP: Although it is well known that X-rays cause mutations, they are routinely used to diagnose medical...

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Similar questions

Although it is well known that X-rays cause mutations, they are routinely used to diagnose medical problems, including potential tumors, broken bones, and dental cavities. Why is this done? What precautions need to be taken?
Why has gene therapy not been the success that was expected when it was first proposed as a viable means of treating genetic disorders?
Several research studies are under way that involve the use of genetherapies to inhibit the growth of cancer cells. Oncogenes are mutant genes that are overexpressed and cause cancer. New gene therapies are aimed at silencing oncogenes by producing antisense RNA that recognizes the mRNAtranscribed from oncogenes. Based on your understanding of antisense RNA , explain how this strategy would prevent the growth of cancer cells.
What's the best definition of gene therapy? a) The introduction of a non-mutated and functional gene into a patient's DNA. b) The delivery of a non-mutated and functional protein into a patient. c) The introduction of a non-mutated and functional gene into a patient's RNA. d) The delivery of drugs that encourage overexpression of a protein to compensate for a mutated protein.
Dr Janis toboggan has two patients who were admitted to her hospital with covid-19. And she gave them both the drug known as remdesivir. Patient A got better and was released from the hospital in two days, but it took a week for patient b to recover and be sent home. That’s interesting though dr toboggan. “Why did the drug work differently in the two patients? Remedesivir seemed to help one patient but not the other. Maybe the drug speeds up recovery but the patients had different levels of infection. Or, maybe remdesvir didn’t have any effect on recovery, and the patients were just at a different places in their infections by the Novel corona virus. To find which hypothesis was a better explanation her data, Dr To boggan applies a little deductive reading. “What if I set up a situation where I have two patients at the same point in their infection and I give one the drug and one not? If remedesivir speeds up recovery, then the one with the drug will recover faster. But if…
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What is the difference between gene therapy and cell therapy?
Give a schematic diagram of how we can Treatment Thalassemia by using gene therapy? Please answer at your own words,please..
Give a schematic diagram of how we can Treatment Sickle Cell Anemia by using gene therapy? Please answer at your own words,please..
1. a)how is it possible for such drugs to selectively kill bacterial cells and not our own cells? b)Provide an example of post-translational regulation of protein activity and explain the advantage of regulating each protein/process at the post-translational level instead of the transcriptional level.
Compare gene therapy with use of RNAi as genetic medicines.