1. a)how is it possible for such drugs to selectively kill bacterial cells and not our own cells? b)Provide an example of post-translational regulation of protein activity and explain the advantage of regulating each protein/process at the post-translational level instead of the transcriptional level.

In the presence of tryptophan, and a mutation in the allosteric domain that abolishes tryptophan binding______. a) the active repressor cannot bind trp), so operon gene transription is attenuated b) the inactive repressor cannot bind trpO, so operon gene transcription occurs] c) the active repressor binds trpP, so operon gene trasncription is repressed, the inducer cannot bind trpO, so gene transcription occurs d) the repressor binds to the corepressor, and an operon gene transcription occurs

A mutation takes place that switches a codon from ACG to ACA. What type of mutation is this? A) nonconservative missense B) silent C)nonsense D) Conservative missense

Can I get help with these questions, please.

What is the effect of a mutation in the allosteric site of the Lac Repressor protein? A) The Lac Operon would always be expressed whenever glucose is absent. B) The Lac Operon would never be expressed in any environment. C) The Lac Operon would be regulated normally. D) The Lac Operon would be never be expressed in lactose but would always be expressed in glucose.

Explain how oseltamivir (Tamiflu) interferes with influenza virus replication.

Answer these questions concerning promoters. a) What role do promoters play in transcription? b) What is the common structure of bacterial promoter with respect to consensus sequences? c) Eukaryotic promoters are more variable than bacterial promoters.  Why? d) What is the meaning of the term alternative promoter? How does the use of alternative promoters affect transcription?

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Answer these two multiple choice questions

Explain the effect of lacI− mutations?