Human Physiology: An Integrated Approach (8th Edition)
8th Edition
ISBN: 9780134605197
Author: Dee Unglaub Silverthorn
Publisher: PEARSON
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Textbook Question
Chapter 7.3, Problem 8CC
Which insulin release pathway in Figure 7.7b is a simple endocrine reflex? Which is a complex endocrine reflex? Which is a combination neural-endocrine reflex?
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Chapter 7 Solutions
Human Physiology: An Integrated Approach (8th Edition)
Ch. 7.1 - Name the membrane transport process by which...Ch. 7.1 - What is the suffix in a chemical name that tells...Ch. 7.2 - What is the classic definition of a hormone?Ch. 7.2 - Based on what you know about the organelles...Ch. 7.2 - Prob. 5CCCh. 7.2 - The steroid hormone aldosterone has a short...Ch. 7.3 - In the blood glucose example, the increase in...Ch. 7.3 - Which insulin release pathway in Figure 7.7b is a...Ch. 7.3 - Prob. 9CCCh. 7.3 - Catecholamines belong to which chemical class of...
Ch. 7.3 - What intracellular structure is used for transport...Ch. 7.3 - Prob. 12CCCh. 7.3 - Match the general reflex pathway patterns shown in...Ch. 7.3 - Prob. 14CCCh. 7 - The study of hormones is called _____.Ch. 7 - List the three basic ways hormones act on their...Ch. 7 - List five endocrine glands, and name one hormone...Ch. 7 - Match the following researchers with their...Ch. 7 - Put the following steps for identifying an...Ch. 7 - Prob. 6RQCh. 7 - Prob. 7RQCh. 7 - Prob. 8RQCh. 7 - Prob. 9RQCh. 7 - Prob. 10RQCh. 7 - Prob. 11RQCh. 7 - Why do steroid hormones usually take so much...Ch. 7 - Prob. 13RQCh. 7 - Prob. 14RQCh. 7 - Prob. 15RQCh. 7 - Prob. 16RQCh. 7 - In reflex control pathways involving trophic...Ch. 7 - What characteristic defines neurohormones?Ch. 7 - Prob. 19RQCh. 7 - Prob. 20RQCh. 7 - Prob. 21RQCh. 7 - Prob. 22RQCh. 7 - Prob. 23RQCh. 7 - Prob. 24RQCh. 7 - Compare and contrast the three chemical classes of...Ch. 7 - Map the following groups of terms. Add terms if...Ch. 7 - The terms specificity, receptors, and...Ch. 7 - Prob. 28RQCh. 7 - Some early experiments for male birth control...Ch. 7 - Prob. 30RQCh. 7 - The following graph shows plasma TSH concentration...Ch. 7 - Prob. 32RQ
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Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- What happens to the insulin-secreting capacity of a type 2 diabetic placed on insulin therapy earlier than recommended? Can the external supply of insulin improve the functional capacity of the insulin-secreting cells, to some extent by providing some rest to these cells? Is inhaled insulin a suitable substitute for injectable insulin? Is there, or will there soon be, insulin in the form of a tablet?arrow_forwardDescribe the relationship between Insulin Growth Factor (IGF) and aging. What role does IGF play in the process of Aging and Longevity? How is IGF involved in signaling for this process? Is the signaling by IGF long term signaling or short term signaling with regard to the interactions. Which other molecule(s) might contribute to duration of IGF signaling for this process?arrow_forwardFor our patient John with diabetes type II, he can make insulin but his body cells can be resistant to this insulin. Note: activated insulin receptors will activate glucose receptors. How is John’s diabetes related to the endocrine system?arrow_forward
- Name two proteins that are effectors of the insulin signaling pathway in adipocytes, liver, or muscle cells. Explain how these effector proteins address the conditions that triggered insulin release.arrow_forwardA woman affected by type I (insulin-dependent) diabetes miscalculates and injects herself with too much insulin. She soon begins to feel confused and sluggish. She calls for medical assistance and injects herself with the glucagon her doctor prescribed for such an emergency. When an ambulance arrives, she is given dextrose ( a sugar) intravenously. How did the excess insulin cause her symptoms. How did the glucagon injection and the intravenous sugar help reverse them?arrow_forwardFor our patient John with diabetes type II, he can make insulin but his body cells can be resistant to this insulin. Note: activated insulin receptors will activate glucose receptors. John takes a drug called metformin, in conjunction with regulating his diet and exercising. This drug has two main effects. First, it helps reduce hepatic (liver) production of glucose. Second, it helps cells become more sensitive to insulin. Explain how these two effects of metformin can help John.arrow_forward
- During an in vitro experiment, it is found that glucocorticoids inhibit the growth of all neural processes, therefore controlling the morphologic characteristics of the chromaffin cells of the adrenal medulla. These modified neurons are most likely stimulated by which of the following mechanisms to release their secretory product? A) Acetylcholine release from sympathetic fibers B) Change in the rate of polymerization of metanephrine C) Decreased blood flow in cortical sinusoidal capillaries D) Downregulation of corticotropin-releasing factor E) Increased concentrations of endogenous chromogranins.arrow_forwardIn patients with type 2 diabetes, cells no longer respond to the hormone insulin. What change has likely occurred in these cells to make them insulin resistant?arrow_forwardHow could the origin of endocrine control systems lie in ordinary cellular events? How might the earliest multicellular organisms have evolved some sort of endocrine coordination?arrow_forward
- what are TRH and TSH? Why are they referred to as tropoc hormones?arrow_forwardUnder what conditions would insulin be released? Is this a hormonal or humoral factor?arrow_forwardSuppose that the circulating concentration of hor-mone is 10–10 M and the Kd for binding to its receptor is 10–8M. What fraction of the receptors will have hormone bound?If a meaningful physiological response occurs when 50% ofthe receptors have bound a hormone molecule, how muchwill the concentration of hormone have to rise to elicit aresponse? The fraction of receptors (R) bound to hormone(H) to form a receptor–hormone complex (R–H) is [R–H]/([R] + [R–H]) = [R–H]/[R]TOT = [H]/([H] + Kd).arrow_forward
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