ANATOMY+PHYSIOLOGY
4th Edition
ISBN: 9781260265217
Author: McKinley
Publisher: RENT MCG
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Textbook Question
Chapter 19, Problem 3CSL
Your grandfather was told that his SA node (pacemaker) has stopped functioning. Explain how his heart is still beating at a rate of 40 to 50 times per minute. Are the atria stimulated to contract? Explain.
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Your grandfather was told that his SA node (pacemaker) has stopped functioning. Explain how his heart is still beating at a rate of 40 to 50 times per minute. Are the atria stimulated to contract? Explain.
1) If a heart lacked gap junctions, an organ-level contraction would occur.True/False
2) We care about mean arterial pressure specifically because we care about having an appropriate rate of blood flow through capillaries. True/False
3) A person's L-type channels are mutated so that they do not inactivate. How would this affect the time duration of a cardiac action potential?
Group of answer choices
a)Increase
b)No change
c)Decrease
4) A person's L-type channels are mutated so that they do not inactivate. How would this affect the time duration of a cardiac muscle cell contraction?
Group of answer choices
a)Increase
b)No change
c)Decrease
John is your 68-year old neighbor who was recently told by his doctor that his heart’s main pacemaker stopped working (“They told me it is called an SA node”). He knows you are a nurse, so he came to you to ask why his heart is still beating, but only at a rate of 50 times per minute. Please explain using appropriate physiological and anatomical details.
Chapter 19 Solutions
ANATOMY+PHYSIOLOGY
Ch. 19.1 - Prob. 1WDYLCh. 19.1 - Prob. 2WDYLCh. 19.1 - Prob. 3WDYLCh. 19.1 - Prob. 4WDYLCh. 19.1 - Prob. 5WDYLCh. 19.2 - What is the bony structure that protects both the...Ch. 19.2 - Prob. 7WDYLCh. 19.2 - Prob. 8WDYLCh. 19.3 - Prob. 9WDYLCh. 19.3 - Prob. 10WDYL
Ch. 19.3 - Prob. 11WDYLCh. 19.3 - Prob. 12WDYLCh. 19.3 - Prob. 13WDYLCh. 19.3 - Prob. 14WDYLCh. 19.3 - Prob. 15WDYLCh. 19.3 - Prob. 16WDYLCh. 19.3 - What areas of the heart are deprived of blood when...Ch. 19.3 - Prob. 18WDYLCh. 19.4 - Prob. 19WDYLCh. 19.5 - Prob. 20WDYLCh. 19.5 - Which autonomic division is associated with the...Ch. 19.6 - Prob. 22WDYLCh. 19.6 - What is autorhythmicity? Describe how nodal cells...Ch. 19.6 - What is the path of an action potential through...Ch. 19.6 - What anatomic features slow the conduction rate of...Ch. 19.7 - In which direction does Ca2+ move in response to...Ch. 19.7 - What three electrical events occur at the...Ch. 19.7 - What is the significance of the extended...Ch. 19.7 - What events in the heart are indicated by each of...Ch. 19.8 - Pressure changes that occur during the cardiac...Ch. 19.8 - What is occurring during ventricular ejection?Ch. 19.8 - Prob. 32WDYLCh. 19.8 - Define end-diastolic volume, end-systolic volume,...Ch. 19.9 - What are the two factors that determine cardiac...Ch. 19.9 - What is the cardiac output at rest and during...Ch. 19.9 - Prob. 36WDYLCh. 19.9 - Describe the atrial reflex, which involves...Ch. 19.9 - Prob. 38WDYLCh. 19.9 - Prob. 39WDYLCh. 19.10 - What would be the path of blood flow through the...Ch. 19 - Which of the following is the correct circulatory...Ch. 19 - The pericardial cavity is located between the a....Ch. 19 - How is blood prevented from backflowing from the...Ch. 19 - ____ 4. Venous blood draining from the heart wall...Ch. 19 - _____ 5. Calcium channels in the nodal cells...Ch. 19 - ____6. Action potentials are spread rapidly...Ch. 19 - Why is it necessary to stimulate papillary muscles...Ch. 19 - ____ 8. Preload is a measure of a. stretch of...Ch. 19 - ____ 9. All of the following occur when the...Ch. 19 - ____10. What occurs during the atrial reflex? a....Ch. 19 - Prob. 11DYKBCh. 19 - Compare the structure, location, and function of...Ch. 19 - Prob. 13DYKBCh. 19 - Explain why the walls of the atria are thinner...Ch. 19 - Describe the structure and function of...Ch. 19 - Explain the general location and function of...Ch. 19 - Describe the functional differences in the effects...Ch. 19 - Prob. 18DYKBCh. 19 - List the five events of the cardiac cycle, and...Ch. 19 - Define cardiac output, and explain how it is...Ch. 19 - A young man was doing some vigorous exercise when...Ch. 19 - A young man was doing some vigorous exercise when...Ch. 19 - Prob. 3CALCh. 19 - Prob. 4CALCh. 19 - During surgery, the right vagus nerve was...Ch. 19 - Prob. 1CSLCh. 19 - Prob. 2CSLCh. 19 - Your grandfather was told that his SA node...
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- Your grandfather was told that his SA node (pacemaker) had stopped functioning. Explain how his heart is still beating at a rate of 40-50 times per minute. Are the atria still being stimulated to contract, and what area of the heart is stimulating them?arrow_forward1)An increase in total peripheral resistance, all else remaining the same, will cause an increase in mean arterial pressure.True/False 2) We care about mean arterial pressure specifically because we care about having an appropriate rate of blood flow through capillaries. True/False 3) A person's L-type channels are mutated so that they do not inactivate. How would this affect the time duration of a cardiac action potential? Group of answer choices a)Increase b)No change c)Decrease 4) A person' L-type channels are mutated so that they do not inactivate. How would this affect the time duration of a cardiac muscle cell contraction? Group of answer choices a)Increase b)No change c)Decreasearrow_forwardConsidering the function of the heart, why is it important that the myocardium cannot be tetanized?arrow_forward
- Although not considered the primary pacemaker of the heart, the atrioventricular node can indeed be considered as playing SOME role in setting the rhythm of the heart (kind of a secondary pacemaker). Explain how/why.arrow_forwardRoberto is a 78-year-old male who has recently been diagnosed with Class III Congestive Heart Failure (CHF). His physician told him that his left ventricle has become stiff and cannot relax. Although there is no current treatment that can “fix” Roberto’s condition, he is prescribed two medications: an ACE inhibitor and beta blockers. Briefly describe how beta-blockers will influence Roberto’s heart function, specifically the physiology of the ventricles.arrow_forwardRoberto is a 78-year-old male who has recently been diagnosed with Class III Congestive Heart Failure (CHF). His physician told him that his left ventricle has become stiff and cannot relax. Although there is no current treatment that can “fix” Roberto’s condition, he is prescribed two medications: an ACE inhibitor and beta blockers.Briefly describe how beta-blockers will influence Roberto’s heart function, specifically the physiology of the ventricles.arrow_forward
- Why do we need to prevent the decompensated state in patients with heart failure?arrow_forwardDescribe the events leading up to contraction in a cardiac contractile cell. (Where does the stimulus to “contract” come from?)arrow_forwardWhen the SA node doesn’t function properly, the AV node takes over pacing the heart and produces what is known as a junctional rhythm. Explain why we don’t see P waves on the ECG of an individual with such a rhythm?arrow_forward
- You wanted to prepare a fish dish for your grandfather that he loves the dried salted Codfish. But you noticed that this might be causing a problem for your elder grandfather since he has hypertension, what would you do to remove the salt from the fish? (Answer with one word) What is the mechanism by which the salt gets out from the fish? (one word) Please put a comma and space between * the two answers word. Answer:arrow_forwardThe exact cause of blood clotting in the left atrium of the heart during atrial fibrillation (AF) is not known. In part it might be due to blood stasis (not moving) or it may result in combination with endothelial cells damage. (A) What are the potential mechanisms that might drive clotting and what markers might you look for in the blood to predict potential risk? (B) Other than treating the arrhythmia, what other pharmacological intervention might be provided to AF sufferers?arrow_forwardDescribe why a doctor would give a patient that is diagnosed with acute myocardial infarction the treatment of tissue plasminogen activator(tPA)? What will it do?arrow_forward
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