Why can acetylcholine (or other signaling molecules) have so many different effects?

Which is a correct explanation of an aspect within G protein signaling pathway? ATP powers the G protein to cause it to move and activate an enzyme O GTP activates the G protein causing it to move to activate the next protein O The receptor is active when bound to GTP GTP causes the G protein to activate and bind to the receptor

Which of the following statements is true?   Question 2 options:   a)  All cells will respond to the hormonal signal because hormones are broadcast throughout the body.   b)  The regulation of inflammatory responses at the site of an infection is an example of paracrine signaling.   c)  Paracrine signaling involves the secretion of stimuli into the bloodstream for distribution throughout the organism.   d)  The axons of neurons typically signal target cells using membrane-bound signaling molecules that act on receptors in the target cells.

An extracellular signaling molecule binds to cell surface receptor. This cell surface receptor changes shape and activates a trimeric G-protein.  What kind of receptor is this? a) Ion-channel coupled receptor b) Receptor Tyrosine Kinase c) G-Protein coupled receptor d) All of the other answers are correct e) Enzyme-coupled receptor

What's the role of Protein Phosphatase 1 (PP1) in cell signaling?

Briefly explain how integrin inside-out and outside-in signaling is different structurally and functionally? (hint: explain what happened to ligand binding and conformation)

What is the difference between a protein kinase and a secondmessenger? Can both operate in the same signal transduction pathway?

Compare three types of signaling molecules: neurotransmitters, hormones, and local regulators.

Describe the difference between agonist and antagonist. What is a role of    G-protein coupled receptors?

Although insulin initially acts through a tyrosine kinase receptor it also subsequently results in activation of a Ser/Thr protein kinase. Which one? A) Protein kinase A B) Protein kinase B C) Protein kinase C D) Ras E) Phosphoinositide-3-kinase