Why are dominant mutations in proto-oncogenes generally not inherited? A. Inherited mutations in proto-oncogenes would not affect cell division rate B. Inherited mutations in proto-oncogenes would decrease cell division rate and no viable embryo would form C. Inherited mutations in proto-oncogenes would increase cell division rate and no viable embryo would form D. Dominant mutations in proto-oncogenes are always inherited

Why are dominant mutations in proto-oncogenes generally not inherited? A. Inherited mutations in proto-oncogenes would not affect cell division rate B. Inherited mutations in proto-oncogenes would decrease cell division rate and no viable embryo would form C. Inherited mutations in proto-oncogenes would increase cell division rate and no viable embryo would form D. Dominant mutations in proto-oncogenes are always inherited

Biology (MindTap Course List)

11th Edition

ISBN:9781337392938

Author:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Chapter33: Plant Structure, Growth, And Development

Section: Chapter Questions

Problem 10TYU: Cell differentiation occurs through _______________, whereas morphogenesis involves _______________....

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What is the difference between a proto-oncogene and an oncogene? a. oncogenes cause benign tumors (polyps), proto-oncogenes cause malignant tumors b. oncogenes transform cells to hyperproliferation, proto-oncogenes have a normal cellular role c. oncogenes have undergone post-translational modification, proto-oncogenes have not d. oncogenes have undergone intron splicing, proto-oncogenes have not
One example of an oncogene is ras. Ras is a G protein that is activated when growth factors bind to a G protein-coupled receptor. The mutated form of ras binds to GTP but does not allow the GTP to be broken down to GDP. Ras is involved in both the G1G1 and G2G2 checkpoints. What happens to cells that have this mutated form of ras? A. They divide when they should not. B. They don’t divide when they should. C. They enter mitosis without going through the S phase of the cell cycle. D. They enter cytokinesis without going through the M phase of the cell cycle.
Match the following statements to the type of gene they describe. ✓Encode proteins that promote cell cycle progression ✓Encode proteins that regulate cell cycle checkpoints ✓Mutations are loss-of-function and recessive ✓Mutations are gain-of-function and dominant ✓RB1 is an example of this type of gene A.proto-oncogene B.tumor suppressor gene
#9) Cancer cells generally have missense mutations in p53 gene, resulting in truncated p53 normally active p53 dominant negative p53 inactive p53 #2) When cancer cells have not spread beyond its original site, the term used to describe it is benign growth intraepithelial neoplasia carcinoma stage 3 carcinoma in sit #10) Single or double stranded breaks in DNA activate Chk 1 and 2 kinases, which phosphorylates p53. This results in --- in the level of p53 in the cell. increase decrease please answer them all. they are very short and won't take your time. Thank you in advance.
Cell differentiation occurs through _______________, whereas morphogenesis involves _______________. (a) pattern formation; cell determination (b) cell division; cell expansion (c) cell expansion; pattern formation (d) cell determination; cell division (e) cell determination; pattern formation
Describe the steps by which the TP53 gene responds to DNA damage and/or cellular stress to promote cell-cycle arrest and apoptosis. Given that TP53 is a recessive gene and is not located on the X chromosome, why would people who inherit just one mutant copy of a recessive tumor-suppressor gene be at higher risk of developing cancer than those without the recessive gene?
1. a)Proteins that stimulate/promote progression through the cell cycle are encoded by (oncogenes or tumor suppressor genes). Boldface one. b)Proteins that inhibit progression through the cell cycle are encoded by (oncogenes or tumor suppressor genes). Boldface one. c)What is the difference between a proto-oncogene and an oncogene? d)To cause cancer, proto-oncogenes require (1 or 2)allele(s) to be mutated and therefore are considered (dominant or recessive). The mutation results in a (loss or gain) of function. For each underlined pair, boldface one. e)To cause cancer, tumor suppressor genes require (1 or 2)allele(s) to be mutated and therefore are considered (dominant or recessive). The mutation results in a (loss or gain) of function. For each underlined pair, boldface one.
Explain why mutations in tumor suppressor genes are recessive (both copies of the gene must be defective for the regulation of cell division to be defective), whereas mutations in oncogenes are dominant.
A set of HeLa cells is being considered for an experiment. When these cells are analyzed using a machine that sorts cells according to the amount of DNA they contain, the graph below is obtained. Which peak, A or B, most likely represents cells in G1 phase?
You are working in a cell biology lab that investigates non-small cell lung cancer cells, which of these cellular features will be suggestive of senescence in the cells observed? Choose all that apply: Group of answer choices Large flattened morphology Reduced incorporation of 5-bromodeoxyuridine (in DNA replication) Increased p53 expression Decreased expression of p15INK4B
describe the relationship between a proto-oncogene and an oncogene, and explain how one arises from the other. Explain how a mutation in the EGF receptor, or in a GTP- binding protein, can lead to unregulated cell division.
I am confused about how stem cell transplants works. If you put a semi differentiated tadpole nucleus in a denucleated egg cell, it’ll develop into a fully formed tadpole, but if you put a fully differentiated tadpole nucleus, it won’t (because the genes have already been expressed in a way where the cytoplasmic determinants cannot operate to the fullest). Then how come in stem cell transplants, you use adult nucleuses that are already developed and transplant them into the denucleated egg cells?