Which of the following DNA repair pathways repair DNA double strand breakage that only occur in dividing cells? Please select ALL correct answer options. Synthesis-dependent strand annealing (SDSA) Double-strand break repair (DSBR) O Nucleotide excision repair (NER) ONon-homologous end joining (NHEJ) O Mismatch repair (MMR)

Which of the following mechanisms does not contribute to reducing the overall in vivo mutation rate found in most species? Mismatch repair of incorrectly inserted bases Replication-associated proteins, in addition to DNA polymerase, aid in fidelity of replication O Excision repair of thymine dimers O Tautomerization of nitrogenous bases Proofreading by DNA polymerase's 3' to 5' exonuclease activity

There are 2 parts to this question: The following DNA strand (below) is about to undergo DNA replication. a) Please replicate the parental strands into two exact copies TC GATATCGG AGCTATAGCC b) place a centromere between the two replicated copies (or tell me where the centromere would be located),

Which of the following would be involved in the excision repair of a thymine dimer? O DNA polymerase I Nuclease Topoisomerase Primase

Can you help find the true statement of this problem?

Which of the following DNA repair pathways repair DNA double strand breakage that only occur in diving cells?. Please select all correct answers. A. Nucleotide excision repair (NER) B. Mismatch repair (MMR) C. Double- strand break repair(DSBR) D.non- homologous end joining (NHEJ) E. Synthesis-dependent strand annealing(SDSA)

Cytosine can be deaminated to form Uracil   What type of mutation is this classified as? Discuss what happens to the base-pairing properties from switching from C to U? When U is replicated in two rounds of synthesis, what substitution does this result in? Before Uracil alters the DNA during replication, what repair system can be used to correct this error? Describe how this type of DNA repair works?

Below is a list of types of repair systems. Please make sure to select ALL of the processes that can repair double-stranded breaks. base-excision repair O mismatch repair homologous recombination Onucleotide-excision repair ONon-homologous end joining

Why do we find multiple, short RNA pieces inserted into the lagging DNA strand during DNA replication?   Question 43 options:   the DNA polymerase is replaced by the RNA polymerase in these regions for unknown reasons   the DNA polymerase doesn't copy flawless and adds sometime uracil instead of thymine to the growing daughter nucleotide strand   multiple RNA primers have been added with the help of the primase enzyme to create multiple start sites for  the DNA polymerase   Retroviruses sometimes like to insert their RNA into the replication fork and favor the lagging strand   multiple RNA pieces are most likely found in the leading not the lagging strand during DNA replication

I'm a bit confused about this question  will someone help me understand the concept