Which of the following cellular concentrations would be likely to increase the rate of glycolysis? Low AMP levels High glucagon levels Low fructose-2,6-bisphosphate levels High AMP levels High phosphoenolpyruvate levels Low ATP levels

The compound glucose 6-phosphate is NOT encountered in which of the following processes? conversion of pyruvate to glucose conversion to glucose to pyruvate conversion of glycogen to glucose conversion pyruvate to citrate conversion of glucose to fructose 1,6-bisphosphate

iodoacetate is an irreversible inhibitor of glyceraldehyde-3-phosphate dehydrogenase. which of the following is the expected outcome of the exposure of a cell to iodoacetate - Cellular resiration will stop if the cell solely depends on glucose as fuel - The rate of cellular respiration will increase - The reoxidation of coenzymes will noy be affected , but the efficiency of oxidative phosphorylation will be diminished  - There will be no effect on cellular respiration as long as the cell has a large glucose reserve  -The cell sill not be able to reoxidize the reduced coenzymes

The net yield of one molecule of glucose completing glycolysis and the TCA cycle is ... Group of answer choices 2 ATP + 2 GTP + 10 NADH + 2 FADH2 + 6 CO2 2 GTP + 8 NADH + 2 FADH2 + 6 CO2 33 ATP + 6 CO2 2 ATP + 2 NADH + 2 pyruvate

Why would LDH be up- or down-regulated depending on location of cancer cells?   Anaerobic cancer cells enhance rates of ATP production and rely on pyruvate to lactate conversion. LDH5 expressed can vary in favoring pyruvate for conversion to lactate (anaerobic metabolism). Aerobic compartments in newly formed fibrobasts and vessels use lactate to convert to pyruvate for oxidative phosphorylation and favor lactate conversion to pyruvate by LDH1 (aerobic metabolism). Lactate dehydrogenase is upregulated in both cancer cells and supporting stroma and vessels, thus only LDH5 is upregulated showing enhanced expression levels and thus presence at the plasma membrane to increase levels of lactate for export, LDH1 levels are always consistent. LDH1 is upregulated in response to the export of lactate by MCT1, while LDH5 is down-regulated Neither LDH1 or LDH5 are both consistently expressed, regardless of oxygen levels

Although both hexokinase and phosphofructokinase catalyze irreversible steps in glycolysis and the hexokinase-catalyzed step is first, phosphofructokinase is nonetheless the pacemaker of glycolysis. What does this information tell you about the fate of the glucose 6-phosphate formed by hexokinase? Glucose 6-phosphate must be unstable and release its phosphoryl group over time. Glucose 6-phosphate must be utilized by other metabolic pathways. Glucose 6-phosphate must allosterically inhibit phosphofructokinase, but not hexokinase. Glucose 6-phosphate must be wasted when it is produced in excess.

Glycolysis occurs in two stages. Describe what is accomplished in each stage.

During glycolysis (under anaerobic conditions), for each glucose molecule broken down, how many ATP molecules are used and how many are made in total? A diagram summarizing the different reactions of glycolysis is given below. ATP ADP ATP ADP Glucose Glucose-6-phosphate Fructose-6-phosphate Fructose-1,6-bisphosphate Dihydroxyacetone phosphate 2 (Glyceraldehyde-3-phosphate) Glyceraldehyde- 3-phosphate 2 (1,3-Bisphosphoglycerate) 2 (3-Phosphoglycerate) 2 (2-Phosphoglycerate) 2 H₂O 2 (Phosphoenolpyruvate) 2 (Pyruvate) 2 NAD+ + 2Pi 2 NADH + 2H 2 ADP 2 ATP -2 ADP 2 ATP 2 ATP molecules are used and 2 ATP molecules are made 2 ATP molecules are used and 4 ATP molecules are made O 6 ATP molecules are used and no ATP molecules are made O 4 ATP molecules are used and 2 ATP molecules are made O No ATP molecules are used and 6 ATP molecules are made

what is one physiological consequence that might occur if gluconeogenesis was the exact opposite of glycolysis?  please help

The glycolytic enzyme glucokinase (in human liver cells) catalyzes the conversion of: glyceraldehyde-3-phosphate into 1,3-bisphosphoglycerate (producing NADH) phosphoenolpyruvate into pyruvate (producing ATP) fructose-6-phosphate into fructose-1,6-bisphosphate (consuming ATP) glucose into glucose-6-phosphate (consuming ATP) 1,3-bisphosphoglycerate into 3-phosphoglycerate (producing ATP)