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- 00 Examine the structures below of several anti-viral drugs. Match the anti-viral drugs with the correct description or mechanism: NH2 HN NH HO. HN H OH N=N=N AZT (Zidovudine) HO HO. Solvadi Amantadine Acyclovir Nevirapine inhibits the viral DNA polymerase of herpesviruses | əsooy) | an allosteric (noncompetitive) inhibitor of HIV reverse transcriptase | Choose ] blocks the M2 channel in the Flu virion so the virus cannot sense acidic pH Amantadine the first nucleoside analog designed to inhibit HIV reverse transcriptase [ Choose ] Hepatitis C drug that blocks the viral RNA polymerase [ Choose ] 31 EEO 24 6 4. 9Explain how Stearic acid works again virus and kill virus? Explain the mechanism of action? Please write at your own easy words.1. Precise words:Find the nonspecific terms in the following sentences. Replace the nonspecific choices with more preciseterms or phrases (It is not necessary to change the sentence structure).(i) All OVE mutants showed enhanced iP concentrations.(ii) Plants were kept in the cold overnight.(iii) To provide proof of concept for our hypothesis, we studied a virus in its host cell.(iv) The present paper reports on continuing experiments that were performed to clarify thissurprising effect.(v) The first transition state is a little lower in energy than the second transition state. 2. Simple words:Improve the word choice in the following examples by replacing the underlined terms or phrases withsimpler word choices (do not change the sentence structure).(i) These data substantiate our hypothesis.(ii) The difference in our results compared to those of Reuter et al. (1995) can be accounted forby the fact that different conditions were used.(iii) For the purpose of discussing cell migration we…
- Considering that each virus must bind to a specific cell surface receptor for attachment, explain how you would create a drug that prevents viral attachment.Please help me with this question. More than one answer may be correct. The graph relating to the information is included below. The above graph is looking at the relative entry of SARS-CoV2 isolated spike proteins into human cells in culture at two times points: before introduction of the virus to the cell culture (0 minute) and 5 minutes after introduction of the virus to the cell culture. CHC stands for "clathrin heavy chain" and siRNA stands for small interfering RNA, which is often used in experiments as an easy method to stop expression of a certain gene. So in this case, the CHC siRNA would stop cellular production of CHC. The control siRNA introduces a small RNA fragment that does not match any gene as a negative control. Imagine that the experiment was then allowed to run for 500 minutes in addition to five minutes? How would the relative uptake of the spike protein into the control siRNA cells compare after 500 min to the 5 minute treatment? Options: 500 min time point would…HPV viruses have a distinct capability to evade confrontation with the human immune system? What are the basic virus properties responsible for this capability? (Give at least 3)
- Please help me with this question. More than one answer may be correct. THe graph relating to the information is included below. The above graph is looking at the relative entry of SARS-CoV2 isolated spike proteins into human cells in culture at two times points: before introduction of the virus to the cell culture (0 minute) and 5 minutes after introduction of the virus to the cell culture. CHC stands for "clathrin heavy chain" and siRNA stands for small interfering RNA, which is often used in experiments as an easy method to stop expression of a certain gene. So in this case, the CHC siRNA would stop cellular production of CHC. The control siRNA introduces a small RNA fragment that does not match any gene as a negative control. From the above figure, you can conclude what about the process that allows the SARS CoV-2 virus entry into the cell? Options: The spike protein is not involved in cell entry, contrary to popular belief This is evidence for clathrin-mediated endocytosis as the…please tell me how bacteriophage and animal viruses multiply. please add both lytic and lysogenic cycles for bacteriophage, all steps, and the outcomes of both. For animal viruses, please include all steps and briefly explain the differences between their nucleic acids. i am having a hard trouble understanding this information if you could please explain in way where a child would understand it that would be amazingCharged spherical viruses have polymer chains attached to their surfaces, e.g. they are ‘PEGelated’ in the language of a synthetic chemist. At what distance of separation does the entropic force due to the chains become significant compared with that due to the inter-virus electrostatic repulsion?
- Can i get a help answering this question: The case study tells you that the HIV virus exits the cell by budding, but how can you tell that just based on the structure of the HIV virus? (see attached for the case study)each virus bind to a specific cell surface receptor for attachment, so prevention of viral attachment how would you create a drug which will prevent viral attachment.You work for a large biotechnology company that is studying viruses, vaccines, and small molecule inhibitors. You are asked to give an overview of cell signaling as it relates to the coronavirus and young adults. a) Describe cell signaling pathways (e.g., at least 3) that are likely manipulated during a coronavirus infection. In your description, include how the pathways are involved in antagonizing the host antiviral response.