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What is Brain-derived neurotrophic factor (BDNF)? What connection does it have to exercise, nature and cellphones?
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- What are neurotransmitters ? Why are they used ?A mutation causes a switch in dopamine receptor G-protein-coupled mechanisms in the basal ganglia, causing D1 and D2 receptors to swap their G-protein subunits. If these animals began showing Parkinson-like neuronal degeneration, what would be the resulting motor function? Question 1 options: A) Hyperkinetic B) No change C) HypokineticI have stimulated a neuron with dopamine for a second followed by treating it with cocaine and calcium chelating agent (which chelates out all calcium from the system making them unavailable for function), what will be the effect of such impulse on generating an action potential in the post-synaptic neuron? Will it be different if I pretreat the set of nerve cells with cocaine and calcium chelating agent followed by stimulating the neuron with dopamine? Justify your answer with proper reasoning in brief.
- Selective serotonin reuptake inhibitors (SSRIs) are drugs that can alleviate symptoms of depression by blocking the reuptake of serotonin (5-HT) from the synaptic cleft, thereby increasing the amount of time that 5-HT remains active. Elevated levels of 5-HT within the synapse are associated with feelings of well-being; conversely, low levels of 5-HT are correlated with depressive symptoms. Recent studies have shown that SSRIs can also mediate their antidepressant effects by increasing brain levels of certain cytokines, including interferon gamma (IFNY). IFNY directly induces the expression of the protein p11 in neighboring neurons, which then interacts with 5-HTR4, a 5-HT transmembrane receptor. Figures 1 and 2 provide information about this interaction. 5-HTR4 protein (% of WT) expression CAMP levels (% change control) from 120T 100+ 80+ 60+ 40+ 20+ 0 MEM TOT Figure 1 5-HTR4 protein expression in plasma membrane-enriched fraction (MEM) of hippocampal lysate and in total hippocampal…Your current LTP experimental protocol is as follows: - Isolate a ‘silent synapse’ with only NMDA receptors in the postsynaptic membrane. - Induce LTP via low frequency presynaptic stimulation coupled with postsynaptic depolarization. You’re able to verify that the synapse has been potentiated, as there are now strong post-synaptic responses to presynaptic stimulation. But you’re unable to prove conclusively that this change was AMPA receptor-mediated. Explain how you will design an experiment to monitor the trafficking of AMPA receptors into the membrane during LTP using the electrophysiological tag, and how this technique works. Explain how your method utilizes 1) Visualization and 2) Electrophysiological recordings to quantify trafficking of AMPA receptors. Provide an example of how electrophysiological data can be interpreted support the hypothesis that LTP induction leads to the trafficking of AMPA receptors into the postsynaptic site.What role do the supporting cells play in neurotransmission ? Describe the role of at least two different types of Supporting cells. Are the supporting cells. as important as the neurons when it comes. to neural Communication ?
- L-Dopa One of the first medications developed to treat Parkinson's disease is a substance called L-DOPA (or Levodopa). This medication can be taken to reduce the motor symptoms of Parkinson's. Unfortunately there is no way to reverse or stop the death of the dopamine producing neurons, but L-DOPA is one of a few treatments that can help manage the symptoms. Your task is to investigate what L-DOPA is and explain why taking L-DOPA might reduce the symptoms of the disease. Write at least one paragraph (5 sentences) to explain your findings in the box below. Use the rubric here to make sure you are explaining the situation thoroughly. This should be entirely in your own words. Do not use phrases from the internet if you do not know what they mean. Rubric (use to check your work) Has major the main points misconceptions not understand of this topic Shows a Understands Clearly does complete understanding How do Neurons 100 85 70 50 use neurotransmitters like dopamine? What do neurons do when…What are the advantages of the second-generation antipsychotics?If given the choice of treating Parkinson’s disease pharmacologically with L-Dopa or invasively using deep brain stimulation which option would you pick and why? (