What is apoptosis? Explain cell signaling pathways that triggers it.

Which proteins are degraded at specific stages of the cell cycle?

Discuss two-three ways that kinases/phosphatases OR proteasomes ensures proper rates of cell division. A- G B I U V C M I Ꭶ C X₂ C x² = 州 순

why do cells contain a genetic program that tells the cell to commit apoptosis?

In other words, the master regulator initiates a program of gene expression that narrow the potential fates of the precursor's cells’ descendants to the type of cells that participate in the organ’s function. Question: Once a master regulator gene is turned on in some cells and not in others, how do cells “remember” that pattern through all the cell division that occur to form a specific organ?

Describe the role of mitochondria in initiating apoptosis?

You are working in a cell biology lab that investigates non-small cell lung cancer cells, which of these cellular features will be suggestive of senescence in the cells observed? Choose all that apply: Group of answer choices Large flattened morphology Reduced incorporation of 5-bromodeoxyuridine (in DNA replication) Increased p53 expression Decreased expression of p15INK4B

Question 10 Review Concept 12.3 Cell Cycle Regulation. Match the term and its description. Each term can only be used once. The control of the sequential events of the cell cycle is [Choose ) termed as Where the cell cycle stops until a go-ahead signal is received [ Choose ] Crowded cells will stop dividing because of this mechanism [ Choose | called The spread of cancer cells to other parts of the body, where [ Choose] they may form additional tumors is called >

Define apoptosis and explain why it is an important process in multicellular organisms. Give specific examples.  own words

Describe the steps by which the TP53 gene responds to DNA damage and/or cellular stress to promote cell-cycle arrest and apoptosis. Given that TP53 is a recessive gene and is not located on the X chromosome, why would people who inherit just one mutant copy of a recessive tumor-suppressor gene be at higher risk of developing cancer than those without the recessive gene?