The sympathetic postganglionic neurons release only norepinephrine while the adrenal medulla secretes mainly epinephrine (80%) and a small amount of norepinephrine (20%). The sympathetic postganglionic neurons do not secrete epinephrine because; Group of answer choices a. Epinephrine is not as widely used in the nervous system as norepinephrine b. Phenylethanolamine-N-methyltransferase (PNMT) which converts norepinephrine to epinephrine is present in the adrenal medulla, but not in sympathetic postganglionic adrenergic neurons c. Acetylcholine which stimulates the secretion of epinephrine does not act on postganglionic neurons d. Dopamine beta hydroxylase in hyperactive in the adrenal medulla

1a.Arrange these ligands in their order of potencies(high to low) 1b. Choose an appropriate concentration of Propranolol needed to block 50% of epinephrine or isoproterenol induced cAMP production via adrenergic receptors expressed in a cell culture model system. 1c. Which of the following has more affinity to bind to adrenergic receptors? And why?I. EpinephrineII. IsoproterenolIII. Propranolol

Please make me understand this question

Which type of adrenergic receptors would promote glycolysis in response to epinephrine/ adrenaline release? a. Alpha-1  b. Alpha-2 c. Beta

Glutamate is Select one: a. All of these b. often synthesized from glial-synthesized glutamine c. an amino acid d. neurotoxic at high concentrations. e. the most commonly used neurotransmitter in the brain

Which of the following is true of the absorption, distribution, and elimination of delta-9-tetrahydrocannabinol?    Question 11 options:   When ingested, THC is quickly and efficiently absorbed, and more than 30 percent reaches the brain within 10 minutes   When smoked, the peak mood-altering and cardiovascular effects occur together, usually within 5-10 minutes   When smoked, THC is rapidly distributed first to the heart and then absorbed into the blood, reaching the brain and the rest of the body   When ingested, the peak mood-altering and cardiovascular effects occur between 40-60 minutes after ingesting

A patient comes into a hospital emergency room sedated and non-responsive. She had been at a college party where she drank many shots of vodka. What is the most likely explanation for her behavioral and physiological symptoms?   A. The alcohol increased the actions of GABA at the GABA receptor chloride channel   B. The alochol increased dopamine and serotonin activity by running the transporters backwards   C. The alcohol blocked the dopamine reuptake transporter   D. the alcohol activated serotinin autoreceptors in the brain   E. none of the above

Chronic stress has been suggested to be a contributing factor to Major Depressive Disorder. Explain the mechanisms by which the nervous system triggers stress responses, giving examples of the structures and molecules involved in these responses. Discuss how chronic stress might contribute to Major Depressive Disorder. Explain and evaluate the evidence supporting a role for chronic stress in the disorder. Use figures and diagrams to illustrate your answer (This is a neuroscience question)

Below are the steps involved in NMDA activation by excess glutamate. Please place them in the correct order. i. Glutamate activates AMPA ii. Impaired re-uptake of glutamate iii. Membrane depolarization iv. Calcium entry into the neuron v. NMDA activation O v> i > i > i > iv O i > i > iii > v > iv O i > v > iii > i > iv O i >i>v> i > iv

Epinephrine and norepinephrine are chemicals that act as both neurotransmitters and hormones. Describe where they are released from, what organs they target and how they play a role in the sympathetic nervous system.