Reuptake would be considered an antagonistic process. Why?     because it involves the movement of neurotransmitter across a membrane     because it can be affected by drugs     because it decreases the amount of time neurotransmitter stays in the synapse     because it requires energy and yet has no effect

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Quick question on the process of movement    Multiple sclerosis is a disease in which the peripheral neurons lose their myelin sheath. In this disease, voluntary movement of the limbs is slowed because;  Pick the ones that are true  the rate of opening of ligand-gated Na+ channels is decreased.   the generation of the action potential is blocked at points along the unmyelinated region due to the absence of voltage-gated channels   the action potential amplitude is degraded at the Nodes of Ranvier of the peripheral neuron.   The Schwann cells are targeted and destroyed by the immune system, affecting saltatory propagation.   the rate of opening of ligand-gated Na+ channels is decreased.

How could we promote rapid or slow signaling for medical purposes? So there are many examples of slow and rapid signaling via coupled receptors. For an example, salivary gland secretion is rapid signaling and cell growth is slow signaling. These pathways could be twisted and used to resolve medical issues in a more natural and less damaging way. Are there any experiments that have or could be done to test out promoting rapid signaling to speed up slow processes such as cell growth in a severe wound? What experiments have or could be done to test out slowing down signaling that is fast like cells responding to their environment negatively (over-active allergies)? Thank you.

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True or false if false please explain

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How does an action potential in the presynaptic neuron trigger a response in the postsynaptic neuron?     The action potential causes the release of signaling molecules from the presynaptic neuron that bind receptors and stimulates changes in gene expression in the postsynaptic neuron     The action potential "jumps" across the synapse and is continued in the postsynaptic neuron     The action potential causes the release of Ca++ ions in the postsynaptic neuron, which bind to ligand-gated channels that begin an action potential in that neuron     The action potential ultimately triggers the release of neurotransmitters from the pre-synaptic neuron that bind to and open ligand-gated channels in the post-synaptic neuron

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