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Recall the four genera of bacteria that cause opportunistic infections and are physiologically similar to Pseudomonas.
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- What bacterial structural target would make an antibacterial drug selective for gram-negative bacteria? Provide one example of an antimicrobial compound that targets this structure.a recent communicable disease outbreak, except COVID-19. For the selected communicable disease outbreak, the following MUST be clearly identified: a) a description of yellow fever b) who was affected c) location of the outbreak d) the When (time) e) Why the outbreak occurred (fully described what was the cause). f) Provide relevant data and appropriate references.What is the genus and species of your bacteria? And What led you to confirm this result and why? Please discuss. ☆My bacteria is Alcaligenes faecalis
- Clinical application: A 44-year-old man with HIV is receiving antibiotics through a intravenous catheter. The antibiotics are to help treat a kidney infection. The patient develops a fever. Subsequent cultures from the patient's blood, the needle tip, and from the insertion site all show growth of an organism with large oval-shaped cells. The cells reproduce by budding. (a) What is your guess about the identity of the pathogen? (b) How do you think the antibiotics may have contributed to this outcome? (c) What do you think the portal of entry was for this pathogen?Differentiate Streptolysin O from Streptolysin S. What titer is significant for streptococcal infection? Discuss the principle behind ASO titration method based on Neutralization reaction.Hypothetically make the Dichotomous key for Micrococcus luteus, Bacillus subtilis, Bacillus, Megaterium, Bacillus cereus, E.coli, Serratia Marcescens, and Enterobacter aerogenes based on colony morphology. Starting from the results of gram staining, catalase tests, (lactose, sucrose and glucose fermentation tests) and other biochemical tests for their accurate identification.
- Identification These bacteria can live in areas with very minimal concentration of oxygen.Consider the photos here which demonstrate antibiotic sensitivities of Staphylococcus aureus strains as determined by the Kirby-Bauer method. Abbreviations are as follows: C = chloramphenicol; CC = clindamycin; CZ = cefazolin; E = erythromycin; NOR = norfloxacin; OX = oxacillin; P = penicillin; RA = rifampin; SAM = sulbactam-ampicillin; SXT = sulfatrimethoprim; TE = tetracycline; VA = vancomycin. Imagine that only two cellular changes occurred in the original strain (the first image, on the top) that resulted in the resistance pattern of the strain in the second image (on the bottom). Which combination of mechanisms could explainthese results?Choose one or more: A.expression of efflux pumps B.overproduction of PABA C.production of β-lactamase D.altered penicillin-binding protein E.modification of either 50S or 30S ribosomal subunits F.altered DNA gyrasepseudomonas dichotomous key. I need to create a "family tree" for the pseudomonas genus. i.e what tests (gram stains, lactose fermentation, etc) differentiate the pseudomonas subspecies apart?
- Polymer nanoparticles, Microparticles and hydrogels have been developed for slow and sustained drug release for slow and sustained drug release application; mark all that apply below: 1)Drug release from hydrogels can be via (a) drug diffusion, (b) degradation of the polymeric matrix and (c) swelling 2) PLGA degrades to lactic acid and glycolic acid, which are not biocompatible 3) Drug release can be triggered through internal and external stimuli - I.e. by use of pH labile chemistries or external triggers 4) Burst release describes the initial faster release rates often observed in drug delivery system, which is then followed by sustained release 5) while burst release is observed with polymer nanoparticles, it is not observed from hydrogelsA bacterium can protect itself against antibiotics in different ways. Describe 4 fundamentally differentmechanisms of antibiotic resistance. Discuss whether there are any basicdifferences or similarities between these 4 mechanisms and, if so, describe them.One of the early results shows that the post-centrifugation pellet of encapsulated cells also contains EA1 and/or Sap. Why is this not proof that Bacillus anthracis cells have both an S-layer and a capsule simultaneously? I need help finding the answer in the article and explain in short answer link to article: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC106848/