Pls help ASAP, thank you! "Which features of glucose metabolism & regulation in the liver are correctly or incorrectly depicted in the figure?" (all answer choices for the drop down menus are: "correct" or "incorrect")
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"Which features of glucose
(all answer choices for the drop down menus are: "correct" or "incorrect")
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- HANDOUT C Glucose-6 Phosphate Dehydrogenase and TPI Deficiencies Glucose-6-P GSH + GSH Reduced glutathione 2 H+ NADP+ H₂O₂ Hydrogen peroxide Glucose-6-phosphate dehydrogenase Superoxide radical NADPH + H+ 6-Phosphogluconolactone GSSG Glutathione Oxidized reductase Glutathione peroxidase Aerobic respiration, drugs, fava beans glutathione 2 H₂O Detoxification SOD: superoxide dismutase GPx: glutathione peroxidase HK: hexokinase GSSG and GSH: glutathione, oxidized and reduced forms, respectively CAT: catalase The diagram above gives important details about the HMP shunt. Superoxides/free radicals dis- rupt membrane lipids, causing changes in membrane permeability. The HMP shunt is expressed in liver cells, red blood cells, and in lipid-producing tissues. 1. Discuss how glycolysis is affected in G6PD patients, using the diagrams to explain your response. 2. How does this effect differ from TPI patients? 3. Why are red blood cells, but not the nervous system, affected in G6PD patients?Alcohol consumption on an empty stomach results in some interesting biochemical as well as embarrassing behavioral alterations. We will ignore the lat t er. Gluconeogenesis falls; there are increases in intracellular ratios of lactate to pyruvate, of glycerol 3- phosphate to dihydroxyacetone phosphate, of glutamate to α - ketoglutarate , and of D-3- hydroxybutyrate to acetoacetate. Hypoglycemia develops rapidly Blood pH also falls. Alcohol consumption by a well - fed individual does not lead to hypoglycemia or alteration in blood pH . (a) Why does ethanol consumption result in the altered ratios? (b) Why does hypoglycemia and blood acidosis result in the hungry individual? (c) Why does the well-fed person not experience hypoglycemia?Glucokinase acts as a glucose sensor in hepatocytes (livercells), a- and b-cells in the pancreas, enterocytes (intestinalwall cells), and the hypothalamus (a control center in thebrain of numerous physiological processes). Explain whyglucokinase can perform this role.
- Y ANTIBODIES DIETARY B12 PERNICIOUS ANAEMIA HYDROXYCOBALAMIN 5. Pernicious anemia is a disease in which the intestine is unable to absorb vitamin B12, frequently due to the loss of the generically named "intrinsic factor" produced by the parietal cells of the stomach lining. Intrinsic factor is a glycoprotein that binds to vitamin B₁2 and is required for its uptake in the ilium. Vitamin B12 is synthesized only by microbes, and we get it either from digesting animal protein or from our commensal microbiome. Vitamin B₁ is unique among metabolic cofactors in that it contains the trace element cobalt, coordinated in a corrin ring structure that is similar to the porphyrin ring system in heme. Loss of intrinsic factor, often through autoimmunity, results in the inability to absorb B12 and ultimately a loss of red blood cells that results in anemia and can be deadly ("pernicious"). ILEUM NUMBNESS (a) In pernicious anemia, N 5-methyltetrahydrofolate accumulates, and cells run out of…During ketosis brought on by a ketogenic diet, liver gluconeogenesis rates are high True FalseGTP or ATP is produced during the conversion of isocitrate into ketoglutarate succinyl CoA into succinate fumarate into malate malate into oxaloacetate
- Comparing Glycolysis Entry Points for Sucrose Sucrose can enter glycolysis by either of two routes: Sucrose phosphorylase: Sucrose + Pi fructose + glucose-1-phosphate Invertase: Sucrose + H20 fructose + glucose Would either of these reactions offer Jin advantage over the other In the preparation of hexoses fur entry into glycolysis?Glucagon secretion inhibits intracellular acetyl-CoA carboxylase activity through a number of different methods. Make a list of everything that comes to mind.In the fasted state, FADH2 and NADH for oxidative phosphorylation are mainly provided by fatty acid oxidation. Explain how fatty acids are mobilised from adipose tissue by the action of glucagon.
- Handwritten Identify the molecule names, enzyme name, enzyme classification and change in reaction(for glycolysis pathway)The Vmax of muscle glycogen phosphorylase is much larger than that of the liver enzyme. Discuss the functional signifi cance of this phenomenon.Triacylglycerols in adipose tissue do not enter the bloodstream as triacyglycerolsexplain