In aerobic conditions in animal cells, oxidative decarboxyliation release CO2 by eliminating the methyl (—CH3) group. : True / False
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1) In aerobic conditions in animal cells, oxidative decarboxyliation release CO2 by eliminating the methyl (—CH3) group. : True / False
2) All enzymes of the cycle are located in the cytoplasm, except succinate dehydrogenase, which is bound to the inner mitochondrial membrane: True / False
3) Malonate is a competitive inhibitor of the enzyme succinate dehydrogenase. In the presence of malonate, one would expect succinate to accumulate: True / False
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- Copper is a cofactor in several enzymes, including lysyl oxidase and superoxide dismutase.Ceruloplasmin, a deep-blue glycoprotein, is the principal copper-containing protein in blood. It isused to transport Cu2+ and maintain appropriate levels of Cu2+ in the body’s tissues.Ceruloplasmin also catalyzes the oxidation of Fe2+ to Fe3+, an important reaction in ironmetabolism. Because the metal is widely found in foods, copper deficiency is rare in humans.Deficiency symptoms include anemia, leukopenia (reduction in blood levels of white blood cells),bone defects, and weakened arterial walls. The body is partially protected from exposure toexcessive copper (and several other metals) by metallothionein, a small, metal-binding protein thatpossesses a large proportion of cysteine residues. Certain metals (most notably zinc and cadmium)induce the synthesis of metallothionein in the intestine and liver.In Menkes syndrome intestinal absorption of copper is defective. How can affected infants…1 a) What is meant by the ATP currency exchange ratio? Why does the oxidation of mitochondrial FADH2 generate one less ATP than oxidation of mitochondrial NADH? b) If 12 H+ are moved across the inner mitochondrial membrane by NADH oxidation, and each ATP synthesized requires 3 H+ to move through ATP synthase, why are only 3 ATP molecules produced by oxidation of each NADH?Describe how a) Mean Arterial Pressure, b) Maximal Oxygen Consumption, and c) Blood Flow are determined using its formula. In addition, you should explain what those factors are including cardiac output, stroke volume, heart rate, resistance, a-vO2 difference, etc. Describe how many ATPs can be generated by complete cycles of beta oxidation of free fatty acid with 20 carbons. You should indicate how many cycles of beta oxidation and Krebs cycle, and total number of products as well
- Cyanide is a chemical that irreversibly binds to (i.e. prevents the functioning of) the enzyme cytochrome oxidase, an important enzyme in the electron transport system. a) Explain, at the cellular level, why cyanide is a lethal chemical. b) Suggest at least one other cellular consequence of cyanide (i.e. what else happens to the electron transport system when cyanide is present), and an explanation of this effect.Listed are some hypothetical medical conditions, describe their effect on cellular respiration (specifically refer to the effect they will have on energy production). a) A person is unable to take glucose into their cells. b) A mutation occurs in the ATP synthase protein and it is inactive. c) The mitochondrial inner membrane is very permeable to H+.Complete the sentence describing the pentose phosphate pathway in cells that require much more ribose 5-phosphate than NADPH. These cells need ribose 5-phosphate but have relatively higher concentrations of NADPH and lower concentrations of NADP*. Choose from the listed words to fill in the blanks: xylulose 5-phosphate, fructose 6-phosphate, glucose 6-phosphate, five, two, three, glyceraldehyde 3- phosphate, erythrose 4-phosphate, sedoheptulose 7-phosphate. One molecule of and two molecules of are used to generate molecules of ribose 5-phosphate by the reverse reactions of the nonoxidative phase of the pentose phosphate pathway.
- Cyanide is a poison that inhibits the electron transport chain by creating a strong and stable bond with Fe–Cu center in cytochrome C oxidase (complex IV). What is the immediate consequence cyanide poisoning? answer choices A.) Prevent oxidation of NADH B.) Prevent reduction of oxygen C.) Prevent reduction of NADH D.) Prevent oxidation of oxygenthis question is about 16 carbon fatty acid A) what are the end procuts of the eight cycles of beta oxidation of a 16 carbon fatty acid? B) assuming 2.5 ATP molecules are produced from one NADH, how many molecules of ATP are produced from the NADH produced during beta oxidation of a 16-carbon fatty acidthe reaction catalyzed by glyceradehyde 3-phosphate dehydrigenase is based on NAD+ and a active site cysteine. Also another phosphate group is added. what is the reason for that? a) because one ATP is consumed b) an inorganic phosphate is activated for ATP synthesis C) NADH can be recycld and than converted back to NAD+ for glycolysis d) because one ATP is generated
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