In a cancer research lab,normal cells have specific biomarker AAP1 while cancer cell display SSR1 biomarker. Which cell biology technique will help you to know the number of cancerous cells as compared to normal cell using this biomarkers?Give details experimental and logical explanation.

Which type of human cancer cells survived most at an RIP concentration of 10 micrograms per liter?   Skin cancer cells   Prostate cancer cells   Breast cancer cells   Liver cancer cells

How does this protein contribute to cell proliferation protein and how would it stop or apoptosis? Protein How would your drug affect the cancer? CDK2 MDM2 P21

In your own words, explain metastasis and why this process is important in the prognosis of a patient, and is this an easy and efficient process for cancer cells, why or why not?

Of the following choices a loss of function mutation would most lukely contribute to cancer onset if the mutant gene codes for a :   telomerase   tumor suppressor protein   ATP synthesis enzyme    RNS binding protein

RG, a 6-year-old child, has been diagnosed with Acute Lymphoblastic Leukemia (ALL). The standard treatment is 6-mercaptopurine (Purinethol), an inexpensive drug that inhibits proliferation and is effective against rapidly proliferating cells like cancer cells. The compound is an antimetabolite that interferes with DNA synthesis. However, about 10% of patients experience life-threatening toxicities. 6-mercaptopurine is metabolized and detoxified by the enzyme thiopurine-S-methyltransferase (TPMT). The most common SNPS correlated with low enzymatic activity are TPMT*3A, *3B, *3C and *2. They cause enhanced degradation of the enzyme. Genotyping reveals that RG has the following genotype: TPMT*3A/*2. Questions: A patient with TPMT*l/*1 should receive what kind of dose? Reduced dose Standard dose Increased dose

Which of the following statements are correct about the ability of cancer cells to form a secondary tumor at a site distant from the primary tumor (select all that apply)?   A. Is possessed by majority of cells in a tumor   B. Most micrometasases fail to become secondary tumor   C. Usually occurs prior to establishment of micrometatstases   D. Ability is acquired along with ability to move through circulation   E. Is often acquired after micrometastases have been estabilished

You are evaluating a patient with Nonpolyposis colorectal cancer and his biopsy pathology report reveals a defect in some TSGs involved in DNA mismatch repair, which of these genes is least likely to be implicated in this patients cancer? Group of answer choices SMAD4 MSH2 MLH1 PMS1

Pick one initial of TNM cancer staging and identify what the different stages are and what they mean.

What specific types of cells do chemotherapy medications target to destroy? cells with cancer specific antigens cells with any random mutations actively dividing cells cells that are undergoing apoptosis