How many ATP are made from one (1) molecule of glucose during anaerobic cellular respiration (AKA fermentation)?

List the four stages of aerobic cellular respiration.  In addition, for each stage list the amount of NADH, FADH, CO2 and ATP made per glucose molecule

Describe how a) Mean Arterial Pressure, b) Maximal Oxygen Consumption, and c) Blood Flow are determined using its formula. In addition, you should explain what those factors are including cardiac output, stroke volume, heart rate, resistance, a-vO2 difference, etc. Describe how many ATPs can be generated by complete cycles of beta oxidation of free fatty acid with 20 carbons. You should indicate how many cycles of beta oxidation and Krebs cycle, and total number of products as well

What metabolic pathways are aerobic and which are anaerobic? What are the advantages and disadvantages to aerobic and anaerobic metabolism?

How many ATPs could theoretically be formed from a glucosemolecule carried through aerobic respiration, and how many areprobably formed?

Medical researchers are studying a patient with the newly discovered u LEKS metabolic syndrome, in which a genetic mutaton prevents NADH produced in the cytosol from being reoxidized by the electron transport chain. Suppose the following nutrient molecule is digested and metabolized by this patient under anaerobic conditions, e.g. during a sudden intense exertion: de HO-CH: -OH Он н. HO H OH How many ATP molecules will uttimately be produced by the catabolism of this molecule? Note: you can ussume the catabohism is as complete as it can be under the conditions given above. How many molecules of pyruvate will be produced during dycalysist How many maleoules of lactate will be produced

Why can the consumption of molecular oxygen indicate the metabolic rate of aerobic organisms?

What is the goal of glycolysis? What do we start with and what do we end with? How much net ATP is produced?

What is the physiological significance of aerobic cellular respiration?

As early as the 1930s, it was known that frog muscles could still contract when glycolysis was inhibited. Where did the ATP come from to drive these contractions?