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- Identify the indicated part of the reflex arc model. NERV OUS S YSTEM reflex arc motor neuron sensory neuron effector O interneuronTOMY & PHYSIOL X QLSdDTK9h0bblWvHhTcbYE8B8bP-hHpSr7jusxp4sPqUlmvOLjA/formResponse * Required NERVOUS SYSTEM: LABELING & IDENTIFICATION LABEL THE FOLLOWING STRUCTURES OF THE NERVE CELL: A LETTER A: 30°C SONY Control music playhaclu auWork 1. Comparison of the conditioned and unconditioned reflexes Table 18. 1 Comparative characteristics of the reflexes No Unconditioned reflex Conditioned reflex 1. Inborn Acquired
- 12:42 1 l 5G Today Edit 12:40 AM If a baseball is coming at your face, you blink FAST in a reflex manner. A reflex is composed of all of the following EXCEPT A neuron that travels within the spine up to the cortex of the brain An afferent neuron incoming to the spinal cord OA motor or efferent neuron exiting from the spinal cord O An effector, which carries out the responseReflex Arc Using the terms provided, label the following illustration of a reflex arc. Dendrite Cell Body Interneuron Gray matter Sensory neuron axon Synapse White matter Spinal nerve Motor neuron axon 3 2 From receptor 8. To skeletal muscle effector 5 Spinal cord 6.10:39 A LTE 2. You put your hand on a hot stove eye. Please explain the flow of information along ALL neural pathways from receiving the stimulus to reacting and becoming aware of your mistake. (Hint: you start first by describing everything that specifically occurs during a reflex along a reflex arc). II
- What central fiber tract connects forebrain and brain stem structures with the hypothalamus? cardiac accelerator nerve medial forebrain bundle dorsal longitudinal fasciculus corticospinal tractWatch this short video (http://openstaxcollege.org/l/facialnerve) to see an examination of the facial nerve using some simple tests. The facial nerve controls the muscles of facial expression. Severe deficits will be obvious in watching someone use those muscles for normal control. One side of the face might not move like the other side. But directed tests, especially for contraction against resistance, require a formal testing of the muscles. The muscles of the upper and lower face need to be tested. The strength test in this video involves the patient squeezing her eyes shut and the examiner trying to pry her eyes open. Why does the examiner ask her to try a second time?PNS -- The Cranial Nerves Observe the 12 paired cranial nerves of the brain using both the models and gross specimens found in the laboratory. Use your textbook to identify the following cranial nerves by name and Roman numeral. What organs do these nerves innervate? What types of signals do they carry? Number Name Type Innervated Organs I. Olfactory nerve S II. Optic nerve S III. Oculomotor nerve M IV. Trochlear nerve M V. Trigeminal nerve B VI. Abducens nerve M VII. Facial nerve B VIII. Vestibulocochlear nerve S IX. Glossopharyngeal nerve B X. Vagus nerve B XI. Spinal accessory nerve M XII. Hypoglossal nerve M
- 44- An animal is suffering from exaggerated segment reflexes is due to a disorder in: a- Lower motor neuron C- Alpha motor neuron b- Upper motor neuron d- Gamma motor neuron1:14 C NERVOUS SYSTEM PATIENT TERMS ASSIGNMENT Identify the medical term for the following patient described signs/symptoms or chief complaints (CC): Patient Complaint List: 1. headache 2. tingling sensation 3. really sleepy! 4. can't sleep 5. passed out 6. can't talk, 7. walking during sleep 8. convulsions 9. can't move 10. can't sit still AA A ructure-uploads.s3.amazonaws.comMake a FDAR GUILLAIN BARRE SYNDROME A 53-year-old man was admitted to the hospital with a 3-h history of left-arm weakness, glossolalia, and right eyelid droop. After admission, his condition suddenly worsened, with quadriplegia, bilateral peripheral facial palsy, bilateral ophthalmoplegia, and other neurological symptoms. Based on the findings from a neurological examination, MRI, cerebrospinal fluid analysis, and nerve conduction study, a diagnosis of GBS was made. He received intravenous immunoglobulin (0.4 kg/day) for 5 days. After 20 days of systematic therapy, his dysphagia, dyspnea, facial paralysis, ocular movement disorder, and leg weakness recovered almost completely, but his arms were still moderately impaired, with a power of 4/5. Fortunately, the patient recovered well without any sequelae after 2 years of follow-up. Conclusions: In patients with an atypical presentation, the diagnosis of GBS is often delayed. With this case report, we intend to highlight the fact that…