Could somone explain exactly what this means below? NOT HW just trying to get a better understanding on what this experiment is about.  To produce mice that are deficient for RAC1 in macrophages, female C57BL/6 mice homozygously expressing the floxed Rac1 gene (Rac1fl/fl) [6] were crossbred with male mice homozygously expressing Cre under the monocyte-specific lysozyme M promoter (LC) [12]. Mice were genotyped for Rac1 deficiency as previously described

Can I get help on creating a concept map for the Introduction provided below? The development of a multicellular organ (such as the brain) from stem cells involves complicated processing.1,2 Proliferating stem cells undergo the fate decision process before developing into tissues or organs. This process is controlled and regulated by a signaling pathway network (called gene regulatory network).3 The network comprises pathways created by the intrinsic and extrinsic factors of the cells, which form the signal transduction to control stem cell fate. There are various signal pathways that have been discovered and studied for tissue development and patterning, including Wnt signaling,4 Notch, and receptor tyrosine kinase.5–7 It has been suggested that cross-regulatory interactions between elements of the pathways (e.g., Wnt and Notch) affect the process of cell fate assignment.8,9 Most components of Wnt signaling have multiple interactions with other signaling pathways (e.g., Hippo/YAP and…

Solve the whole question give in photo.

The following questions are based on information given below: Puc is a transcriptional target of the JNK signaling pathway. Therefore, the level of Puc-LacZ reporter can be used to monitor JNK signaling activity. Ubi-GFP is a construct with GFP expression under the control of a ubiquitously expressed promoter. To study the effect of loss of dmyc function during wing disc development, you used the dmycP0, which is a partial loss of function allele, and generated the following genotype flies: dmycP0FRT19 / Ubi-GFP FRT19; +; Puc-lacZ / Hs-FLP.    After heat shock to induce mutant clones, you examined wing discs for GFP and Puc-LacZ levels (shown in green and red in the images below, respectively) 1. Identify and mark the cells that are homozygous wild type, heterozygous, and homozygous for the dmycP0 mutation in image A.    1. What effect does loss of dmyc have on JNK signaling levels? Is this effect cell autonomous?