adrenaline adenylyl cyclase CYTOSOL GDP ATP dissociated R-subunit of PKA CAMP inactive PKA active РКА phosphorylase kinase glycogen phosphorylase GLYCOGEN G1P

A certain type of tumor results in the overproduction of glucagon. Researchers claim that treatment with insulin can counteract the effects of the excess glucagon on the pathway shown in Figure 2. Provide reasoning to justify the researchers' claim.

I don't understand, can I get help? I have attached a picture of everything that will be needed to be annotated. Numerous signaling molecules work together in order for glucagon to induce the regulated release of glucose into the bloodstream. Construct an annotated list of each of these signaling molecules (as learned in the lecture video). For your annotated list, please use the format below. You will need to use this format for each molecule in the pathway that we learned.  Name of Molecule:Type of Molecule:Is activated (or inhibited) by:Activates (or inhibits):

Label the molecules in this figure showing how IP3 and DAG function as second messengers. Note: not all labels are used. IP3 and calcium activating protein kinase C Activated phospholipase C DAG triggering calcium release Activated G-protein with GTR bound Siy IP3 triggering calcium release Phosphatidyl inositol Ligand bound to receptor DAG and calcium activating protein kinase C Activated G-protein with GDP bound Copyright The McGraw-Hill Companies, Inc. Permission required for reproduction or display. TH ,,