Administration of insulin decreases the blood concentration of ketone bodies in patients with diabetic ketoacidosis by which of the following mechanisms? A) Decreasing formation of ketone bodies in skeletal muscle B) Decreasing lipolysis in adipose tissue C) Increasing hepatic activity of carnitine acyltransferase D) Increasing hepatic levels of acetyl CoA E) Increasing hepatic oxidation of ketone bodies

A client with Type 1 diabetes mellitus is admitted to the hospital in a semi-conscious state with fever, nausea, and vomiting. The urine sample was positive for ketone bodies. Which of the following statements regarding the client is correct? Question 80 options: a)  Glucagon should be administered to stimulate glycogenolysis and gluconeogenesis in the liver  b)  An injection of insulin will decrease ketone-body production c)  The client should be started on a glucose infusion to help them regain consciousness  d)  The client's blood glucose level is well below 4.4 mmol/L

What is the reason that amino acid degradation spikes approximately 24 hours after starting a fast? (A) The brain adapts to using ketone bodies, and amino acids serve as the primary precursors for ketone bodies.(B) Red blood cells begin using amino acids to drive their citric acid cycle.(C) Amino acids are broken down and used to replenish glycogen. (D) The liver needs to make glucose for brain and RBCs, and amino acids provide the carbon skeleton for gluconeogenesis.(E) The ammonia released from amino acid breakdown is a substrate for b-oxidation, which increases during prolonged fasting.

All of the following are true with respect to alpha-glucosidase inhibitor, except:A. It inhibits intestinal alpha-glucosidase B. It decreases enzymatic conversion of oligo- to monosaccharidesC. It lowers postprandial glucoseD. It causes gastrointestinal disturbances.E. It may cause development of hypertension.