4.0 Basal Dopamine (-)-Norepinephrine (-)-Epinephrine 3.5 3.0 2.5 2.0 B 9 -8 -7 -6 -5 -4 Log [Agonist] (M) Oocyte cyclic AMP levels (pmoles/oocyte) 看 市
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![4.0
Basal
Dopamine
(-)-Norepinephrine
()-Epinephrine
3.5
3.0
2.5
2.0
B.9-8 -7
-6
-5
-4
Log [Agonist] (M)
Oocyte cyclic AMP levels (pmoles/oocyte)
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- What is the maximum allowable dosage of the most commonly used anesthetic solution i.e. 2% Lidocaine HCl with 1:100,000 epinephrine for a 52 kg healthy adult?A 10-week-old child was admitted to hospital suffering from a salt wasting crisis. Ultrasound scans of the patient's adrenal glands revealed they were enlarged but all other major organs appeared to be within normal parameters. Although the child appeared phenotypically female, karyotyping showed the child to be 46XY. Additional testing showed an accumulation of cholesterol esters in the adrenal glands, and it was also noted that the concentrations of ∆4-androstenedione were barely detectable. Genetic analysis of all the enzymes involved in steroid biosynthesis did not detect any detrimental mutations affecting their functionality. Treatment included glucocorticoid and mineralocorticoid administration. QUESTION: State which protein is the most likely to be deficient in this scenario and briefly explain your rationale.Epilepsy is often treated with a combination of drugs such as carbamazepine and phenytoin (polytherapy). CYP3A4 is induced by phenytoin and carbamazepine. Carbamzeipine is cleared by CYP3A4, but phenytoin is not cleared by CYP3A4. What problems might you expect if (a) carbamazepine is removed or (b) phenytoin is removed from this two-drug regimen.
- Dexamethasone 1 Dexamethasone is a glucocorticoid administered as an anti-inflammatory agent and immunosuppressant. It is structurally similar to cortisol produced naturally by your adrenal glands. It treats a number of conditions including inflammation, arthritis, autoimmune conditions, skin and eye conditions, asthma, multiple sclerosis, blood or bone marrow disorders, different types of cancer (e.g. leukemia and lymphoma), and severe allergies. This medication is also administered to patients with severe COVID-19 symptoms to decrease mortality rates*. Given that it is a steroid, which family of macromolecules does dexamethasone belong to? *research from the University of Oxford, "Randomised Evaluation of COVID-19 Therapy" trial, recovery.net 2 Given the structures of dexamethasone (left) and cortisol (right) below, and that they both bind to the same receptor, would you expect the binding of dexamethasone to be to the active or allosteric site of the receptor? 3 Based on the…All the B - adrenergic receptors are linked with Gs G-protein. Physiological processes mediated by the intracellular second messenger cyclic AMP include: YOU CAN CHOOSE MORE THAN ONE A.carbohydrate breakdown by the liver (glycogenolysis) B.increased contractility (e.g. increased cardiac contractility) C.smooth muscle relaxation (e.g. bronchodilation or vasodilation) D.Triglyceride breakdown (lipolysis)Catecholamines like epinephrine and norepinephrine among having many physiological effects like increased blood pressure, on a molecular level will also promote the breakdown of what?
- 56. An injection contains 0.50% (w/v) of lido- caine hydrochloride and 1:200,000 (w/v) of epinephrine. Express the concen- tration of lidocaine hydrochloride as a ratio strength and that of epinephrine as a percentage. 56. 1:200 w/v lidocaine hydrochloride 0.0005 % w/v epinephrineBuprenorphine is a partial agonist at mu opioid receptors and is used for opioid maintenance treatment (OMT) in patients with opioid use disorder (OUD). Buprenorphine is formulated in a product that contains the mu opioid receptor antagonist, naloxone. What is the purpose of naloxone in this formulation? O To increase the magnitude of response to the partial agonist. O To reduce the likelihood that buprenorphine will be injected intravenously. O To delay the response to buprenorphine. O To provide pain relief to the patient who has OUD.What type of interactions can glutamic acid participate in that are not possible forglutamine? Explain your answer.
- Discuss how diabetic ketoacidosis (DKA) occurs and its clinical manifestations. In your response be sure to address the following points (i) the three events that characterize DKA; (ii) the underlying molecular causes of each of the three events; (iii) the clinical manifestations and treatments of DKA.All of the following hormones are properly classified EXCEPT (use the table below for general characteristics) (A) thyroxin-arachidonic acid (B) thromboxanes-arachidonic acid derivatives (C) estrogen-steroid (cholesterol based) (D) growth hormone-small protein (< 200 amino acid) (E) norepinephrine- amino acid derivativeTRUE or FALSE GABA is methylated norepinephrine; together both are considered catecholamines.
