1. A population of wild geese has the following frequency foralleles at the alcohol dehydrogenase locus: ADH+ 0.70,ADH null 0.30. In a population of 1,000 birds, what willbe the expected frequencies of each genotype (and thenumber of birds that have each genotype)?*null non-functional protein=2. A major winter storm decimated to this population of wildgeese reducing its number to 100 birds. Their genotypesare displayed below:Genotype +/++/null null/nullNumber 51 42 7Is this an example of genetic drift? Prove this by using the C-squared statistical test where:c2 = (observed expected)²/(expected)-

Approach to solving the question:Question 1:Genotype Frequencies in a Population of 1,000 BirdsWe…

Answered: 1. A population of wild geese has the…

Human Anatomy & Physiology (11th Edition)

11th Edition

ISBN:9780134580999

Author:Elaine N. Marieb, Katja N. Hoehn

Publisher:Elaine N. Marieb, Katja N. Hoehn

Chapter1: The Human Body: An Orientation

Section: Chapter Questions

Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...

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Notice that when comparing our amino acid sequence to a fruit fly, we have a difference of 29 amino acids. That number jumps drastically when we compare ours to the thesis monkey, which has difference of 1. Why does this make sense?
3. The image below depicts 2,3-bisphosphoglycerate depicted in dark gray, and ß subunits are depicted in light gray. (BPG) bound to hemoglobin. The a subunits are BPG (a) Briefly explain how BPG regulates oxygen transport by hemoglobin and describe the critical chemical properties of BPG and its binding pocket.
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The action of this enzyme found in Beano alpha-galactosidase can also according to many sources be used to remove ____________ from the red blood cells of the B blood group and convert them to the O blood group. Since the _________ molecules (composed of many monomers) are connnected to protein on the membrane and also the cell membrane of the red blood cell itself; therefore these molecules help create cell identity and recognition, and when the are connected to proteins they are known as _________ , but if they are connected to the cell membrane itself they are known as ____________. This is a good question for reviewing the structures on the cell membrane as well. The only difference between the B-blood type and the O-blood type is ________. The following is a diagram of the differences of these molecules that determine blood type. List of answers: disaccharid, proteins ,dehydration synthesis ,nucleotide ,amino acids ,nucleic acids ,lactose ,fructose ,sucrose ,hydrolysis…
How would I create a bar graph using the Hardy -Weinberg Equation data ?
e 1.0 0.5- 0 6. Answer the questions below about hemoglobin and myoglobin. a. Sketch and label the binding curves of Mb, Hb, and Hb below. pº₂ b. In the scenarios below for Hb, indicate what effect they would have on the binding curve and give a brief explanation? Scenario Decrease in the concentration of carbon dioxide (CO₂) Distal histidine (the histidine that aids in molecular oxygen binding) mutation to phenylalanine High heat and extreme pH changes Predict if the normal binding curve of Hb will shift right (Hbt), left (HbⓇ), completely to Mb, or no longer bind oxygen completely? Brief Explanation
4
Do not use Ai
1. Draw the Fisher projection of D-glucose, and from Fisher to Haworth projection of ẞ-D-glucose. Label (R) and (S) configuration for each chiral carbon Fisher projection Haworth projection
2. It is known hemoglobinopathies, whi ch arise due to mutations in one of histidines a- or B- chains binding with the iron in the heme gro up.These mutations stabilize the iron in Fes form. Some drugs (eg. sulfonamides) or chemicals( eg.anilin) also can cause methemoglobinemia. Suggest, how change the properties of Hb in these cas es. For this:1)Describe the structure and function of Hb A as a transport protein. 2) Explain the role ofiron ions in act ive centers of Hb for the binding of O23) How many molecules of Oz can bind mutant Hb, if mutation occurs in a- subunits? 4) Why can ascorbic acid be used to tr eat of such patients.
4. PFK-1 is tightly regulated with multiple potent allosteric regulators. Name three of these regulators and explain how they influence the activity of PFK-1.

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