1 Second Marks mmmmmmmmmmmm Observation 1. What is the Heart Rate? 2. What is the rhythm? Regular or Irregular? 3. How is atrial activity related to ventricular activity? Is there a P wave before each QRS? 4. Are P waves upright and uniform? 5. What is the length of the PR interval? 6. Do all QRS complexes look alike? Diagnosis: 7. What is the length of the QRS complexes? econds 3 Second Mark Interpretation

1 Second Marks mmmmmmmmmmmm Observation 1. What is the Heart Rate? 2. What is the rhythm? Regular or Irregular? 3. How is atrial activity related to ventricular activity? Is there a P wave before each QRS? 4. Are P waves upright and uniform? 5. What is the length of the PR interval? 6. Do all QRS complexes look alike? Diagnosis: 7. What is the length of the QRS complexes? econds 3 Second Mark Interpretation

Anatomy & Physiology

1st Edition

ISBN:9781938168130

Author:Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark Womble

Publisher:Kelly A. Young, James A. Wise, Peter DeSaix, Dean H. Kruse, Brandon Poe, Eddie Johnson, Jody E. Johnson, Oksana Korol, J. Gordon Betts, Mark Womble

Chapter19: The Cardiovascular System: The Heart

Section: Chapter Questions

Problem 11RQ: Which portion of the ECG corresponds to repolarization of the atria? P wave QRS complex T wave none...

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Which portion of the ECG corresponds to repolarization of the atria? P wave QRS complex T wave none of the above: atrial repolarization is masked by ventricular depolarization
Which component of the heart conduction system would have the slowest rate of firing? atrioventricular node atrioventricular bundle bundle branches Purkinje fibers
1. rhythm regular or irregular2. determine the heart rate3. do you have p waves before every QRS4. Measure the PR interval5. Measure the QRS duration6. Are there any early or extra beats7. is there normal r wave progression in V1-V6?8. Are the ST segments elevated or depressed?9. Are there any abnormal Q waves?10. Are the T waves of normal size?11. Are there any U waves?12. Is there evidence of myocardial ischemia, injury, or infarction?13. Name this EKG.
1. is the rhythm regular or irregular 2. determine the heart rate 3. do you have p waves before every QRS 4. Measure the PR interval 5. Measure the QRS duration 6. Are there any early or extra beats 7. is there normal r wave progression in V1-V6? 8. Are the ST segments elevated or depressed? 9. Are there any abnormal Q waves? 10.. Are the T waves of normal size? 11. Are there any U waves? 12. Is there evidence of myocardial ischemia, injury, or infarction? 13. Name this EKG.
2.A. 1. 2. 3. 4. |||| ential (mv) 13. State the channels involved in an action potential in cardiac AUTORHYTHMIC cells, which ion moves across the channel, which direction each ion moves across the channel when the channel opens, the approximate membrane potential at which the channel is fully open. +20 !!| CE - | Channel AaBbCcDdEe AaBbCcD. AaBBCEDE Aal Emphasis Heading 1 Normal lon? Influx or Efflux? 14. Define the term pacemaker potential (=drifting potential) and be able to identify it when given a graph of an action potential in an autorhythmic cell such as the one below. I Membrane Potential at which channel opens or is fully open (mv)
help me
1. Identify and color the following in the ECG image below: three P waves three QRS complexes three T waves three PR segments three ST segments two full TP segments two RR intervals ( ● ● ● (RR interval indicates the time for one heartbeat. The RR interval is measured from the peak of one QRS to the peak of the next QRS). سلہ h
DATA ANALYSIS 1. What electrical and mechanical events correlate to the P wave? The QRS complex? The T wave? 2. During which ECG interval does filling of the ventricles occur? During which ECG interval does ejection occur? What is systole and diastole? 3. Describe the path an action potential travels during one cardiac cycle. 4. Describe the flow of blood through the heart and the subsequent flow through the body. 5. What changes occurred in heart rate as position changes from lying down to sitting up? Describe the physiological mechanisms causing these changes. 6. What percentage of the resting cardiac cycle is systole and what percentage is diastole? How did this percentage change with exercise? Explain.
1. Identify and color the following in the ECG image below: ● three P waves (color blue) ● ● ● ● three QRS complexes (color green) three T waves (color a different blue) three PR segments (color red) three ST segments (color purple) two full TP segments (color pink) two RR intervals (color brown) (RR interval indicates the time for one heartbeat. The RR interval is measured from the peak of one QRS to the peak of the next QRS). ملله
8. Circle the term that does not belong in each of the following groupings. 1. AV valves closed AV valves open. SA node is pacemaker Ventricular systole 2. No P wave 3. Ventricles fill 4. Early diastole Semilunar valves open Ventricular pressure drops 5. Stenotic valve Restricted blood flow Heart sound after valve closes 6. Isovolumetric ventricular systole Semilunar valve closed Ventricular systole AV node is pacemaker AV valves open Semilunar valves open Junctional rhythm Late diastole Isovolumetric relaxation High-pitched heart sound Blood volume unchanging AV valve open
15. Rate: bpm P-P atrial rhythm regular? R-R ventricular rhythm regular? Rhythm: Conduction: All waves present? Yes/No, missing. PR interval: (>5?) minim QRS: (>3 ?) ST segment: normal/elevated/depressed T wave: normal/spiked/inverted Ectopic Beats: PACS? Yes/No PVCs? Yes/No # 15. Interpretation: Yes/No Yes/No
4.4. On the circulatory system, what are the correct statements? PHYSIOLOGY_advanced (OJO) Pacemaker cells of the heart are neurons with a stable resting membrane potential. Pacemaker cells of the sinus node, located at the left atrium, determine the heart beat frequency. Cardiac output is a function of the heart beat frequency and the stroke volume. The Frank-Starling mechanism allows coupling of the venous return with the stroke volume. Within each capillary bed there is a net loss of liquid to the extracellular space; this imbalance depends on the hydrostatic pressure and the colloid osmotic pressure gradients.