Surfactant Lab Report: Gas Exchange In The Respiratory System

In this experiment, we are trying to figure out how many droplets of water a penny can hold while face up. Before we began the experiment, we already knew that water molecules stick together as long as gravity isn't stronger than the water. In the experiment we had water, soapy water, and rubbing alcohol.

There are numerous different challenges that the paramedic will face in attempting to keep an airway patent. These challenges vary from patient to patient depending on their condition. One challenge in keeping a patent airway the paramedic will face is trying to maintain the airway of a trauma patient. Trauma patients make it difficult to maintain an airway due to the traumatic damage, especially if it has affected the face and neck regions.

Surfactant is an essential component for the respiratory system to function properly. Knowing the physiology of surfactant in the alveoli is important to know when learning the structure of the air sacs and how they work. This experiment is designed to make the understanding of surfactant in the alveolar film easier to learn, because it's not very simple. Surfactant is a detergent-like substance produced by the Type II alveolar cells in the walls of the alveoli. Surfactant is produced to reduce the surface tension of the water molecules that primarily compose the walls of the alveoli. For this experiment, we will be using milk and food coloring to represent the water (milk) and gas (food coloring) in

movement of water in tissues and maintain the fluidity of mucus and other secretions [3].

Each bubble of casein has a light negative charge. Negative charges are repelled so the bubbles of casein do not bump into one another. Because acid was added the milk gains a small electrical charge. When small amounts of positive charge are floating in the milk, the bubbles no longer repel so they bump together and stick. Instead of microscopic bubbles of protein we have the curds. The reason that the fat that was extracted sat on top of the water is because water is polar and the fat is

Have you ever wondered why your cereal clusters together after milk is poured or why it sticks to the side of your bowl? Even if you try to pull them apart, they come together again. This is a spectacle that happens every day but we just don’t really notice it. Most people don’t really realize that it’s happening everyday. Using fluid mechanics, we can determine why floating objects come together due to surface tension, viscosity and buoyancy (Grayson, 2014).

Surfactants, when dropped in water, will try to spread across the surface of the water. While spreading along, it is also pushing away from the surface of the water. So when an object is placed onto the water, the surfactant is pushing the object away from the water, (while spreading), causing it to

RDS is caused by a defective or delayed production of surfactant in structurally immature lungs. Surfactant is a complex mixture of phospholipids and proteins secreted by the type

Surfactant is a surface-active substance that is made of lipids and proteins that cover the alveoli and stabilize it and also protect the lungs by reducing the surface tension of pulmonary fluids (the amount of energy that is required to increase the surface of a liquid due to its molecular interaction bonds), that’s why alveoli don’t collapse when gas is exchanged in and out of the body (1). Phospholipids take up 80-85% of surfactant’s weight, and 40-50% is in form of disaturated (dipalmitoylphosphatidylcholine, DPPC). DPPC is in control for the surface tension decreasing the capability of surfactant. Surfactant is also composed of phosphtidylglycerol, phosphatiylethanolamine, sphingolmyelin, and cholesterol. Phospholipids in surfactant

As a newborn makes their entrance to this world from intrauterine life all should go smoothly. The newborn is delivered and is stimulated to breathe and then is followed by inspirations, followed by a cry showing a successful arrival to this world, but not all deliveries go as planned. There are diseases of the respiratory system. The most common disease is Respiratory Distress Syndrome (RDS). The Primary cause of RDS is the underdevelopment of the premature lungs states Perretta, 2015. RDS is caused by a deficiency and immaturity of alveolar surfactant with the anatomical immaturity of the premature infants lungs. The incidence of RDS increases with decreasing gestational age says J. Haitsma, 2010. The biggest factor is surfactant deficiency caused by decreased surface area in the lungs for proper gas exchange and thick alveolar-capillary membranes. “Surfactant replacement therapy for preterm infants with RDS has shown to be a major breakthrough in neonatal medicine,” says J. Wirbelauer and Speer, 2009. It has become routine for the prevention and treatment for infants suffering from respiratory distress syndrome. Surfactant replacement therapy is a life-saving treatment for all neonates showing signs of RDS characterized by surfactant deficiency. By replenishing the lungs with an exogenous surfactant shows

In conclusion, the purpose of this experiment was to show how “[t]he main function of surfactant is to lower the surface tension at the air/liquid interface within the alveoli of the lung. This is needed to lower the work of breathing and to prevent alveolar collapse at end-expiration” (Veldhuizen and Haagsman). Although the blue food coloring moved slowly at first, I did not anticipate it would be forced to submerge and move at its own pace. However, when the surfactant was added, it forced the blue food coloring to move at faster rate pushing up and out to integrate with the rest of the water particles changing the entire contents of the petri dish. Surfactant is important to help breakdown and decrease the surface tension allowing the gases

Surfactant is an essential component for the respiratory system to function properly. This experiment was done to better understand the functioning of a surfactant. The reason why a surfactant is needed is to reduce the surface tension of the water molecules that primarily compose the walls of the alveoli. We used milk and food coloring to represent the water and the gas in the respiratory system. The milk was placed in a petri dish and drops of food coloring were added into the milk at the center of the dish.

Surfactant is a natural production around 30 weeks and is fully sufficient to for a baby to breath normally. Fortunately, there are artificial surfactant that can be given to babies to prevent or get rid of respiratory, distress syndrome (RDS). Babies that respond well require less support from ventilators and lower additional oxygen

Hypothesis: If I place an egg in vinegar, then the outer layer of the egg is going to become slimy and look like rubber. It will also become larger and bubbles will form on and around the egg.

The name of the disease that I have selected to write this disease paper about is acute respiratory distress syndrome. Acute respiratory distress syndrome according to our text book Respiratory Care Principles and Practice by Dean Hess states that acute respiratory distress syndrome is “characterized by the abrupt onset of respiratory distress; associated with severe hypoxemia and diffuse pulmonary opacities on chest radio graph that are not caused by congestive heart failure or volume over load; Pao2 < 200 MMHG.” (Hess, 1287). Acute respiratory distress syndrome is respiratory insufficiency marked by progressive hypoxemia due to severe inflammatory damage causing abnormal permeability of the alveolocapillary membrane. The alveoli fill with fluid, which interferes with gas exchange. ETIOLOGY according to Taber’s medical dictionary, acute respiratory distress syndrome can typically be a result of or from trauma to the lungs for example in situations like near drowning, aspiration of gastric acids, and severe lung infection or systemic disorder. Shock, septicemia, disseminated intravascular coagulation, cardiopulmonary bypass or reaction to blood transfusions. Widespread damage to the alveolocapillary membranes is initiated through the aggregation and activity of neutrophils and macrophages and the activation of complement. Cytokines, oxygen free radicals, and other inflammatory mediators damage the walls of capillaries and alveoli, producing diffuse inflammatory interstitial