Sickle Tissue: A Case Study

Per office visit notes dated 8/7/2016, patient presents for a follow up for neck pain, left elbow pain and bilateral wrist pain. Current medications include Naprosyn EC 500 mg, Tramadol 50 mg, Lyrica 50 mg and Levora-28 0.15/0.03 mg. Patient was recommended retrial of Celexa for her depressed mood related to her chronic

The dosage was to high for the patient because the chart started going over the regular normal level

Vaso-occlusive events are moments of severe pain common in sickle cell patients; it is a result of the adhesion of cells to the endothelial lining. While leukocytes play a role in vaso-occlusive events, erythrocyte activity plays an even bigger role. It was discovered that the receptors on the erythrocytes and the endothelial cell were responsible for the mediation of the adhesion of erythrocytes to the endothelial lining (Stuart; Nagel, 2004). The adhesions of these cells to the cell wall is what leads to inflammatory disease, and certain types of acute chest syndrome.

The medications listed in the video were oxygen, morphine, aspirin, nitrates, beta blockers, ace inhibitors, statins, thrombolytics, and anticoagulants.

This is a 50 year-old male who required inpatient hospitalization due to dizziness, abdominal pain, headache, and nausea after taking atenolol. Mr. A came to the Emergency Department with complaints of some dizziness two-day prior to admission and collapsed several times. For the past 2 weeks, he had not been taking his blood pressure medications due to financial reasons. He was given clonidine and atenolol from his dialysis center while he was unable to afford his normal hypertension medication. He was dialyzed three times a week through his arteriovenous fistula and had not missed any sessions. He also had increased dyspnea on exertion over the past 3 weeks, orthopnea and usually sleeps on several pillows which worsened between his dialysis. His medical history is significant for hypertension, diabetes mellitus type II, morbid obesity and end stage renal disease on dialysis.

medication s :Pradaxa 150mg BID, Pepcid 20mg BID, Lipitor 20mg QD, iron 325 BID coaaar 25mg QD, Metformin 1000MG BID, Coreg 3.125mg Q12h, Lasix 20mg QD,

Proteins are macromolecules made up of many amino acids that play a lot of important roles in our body. In living organisms proteins have various functions. For example enzymes are proteins that are very important to our body. Enzymes are invovled in thousands of chemical reaction wporking by lowering the activation energy therefore speeding up the reactons. Proteins can also function as a messenger, transporter, and also for storage in the body. Proteins are polymers made up of a lot of amino acid monomers. The amino acid on the protein gives the protein its special function or unique property. A phenotype is an observable trait or characteristics on an organism. For example eye color, height, and a size of a bird's beak are all phenotypes,they

NH hospitalization record reveals a medical history of a non-injurious stroke, numerous episodes of sickle cell crisis, acute chest syndrome and chronic asthma. NH is prescribed a daily regimen of medications including a daily dose of 15,00 mg hydroxyurea, 1 mg folic acid for his SCD and 44 mcg of inhaled fluticasone for his asthma. Currently, while suffering from sickle cell crisis, NH is prescribed oxycodone 5mg, Toradol 21 mg IV solution, acetaminophen, and morphine as well as a continuous IV drip of D5 ½ NS, KCL. Due to the opioids and level of pain NH has endured the last 4 days (since beginning of crisis) he is exhausted and considered a fall risk due to his fatigue and reports that he naps off and on throughout the day and only gets out of bed to use the

Sickle cell disease (SCD), a genetic disorder, is an autosomal recessive disease (Brown, 2012). This means that two copies of the mutated gene, one from each parent, must be present for their child to develop the disease. If both parents have the sickle cell gene, there is a 50% chance that any one child will also have the sickle cell trait or gene. Also, if both parents have the sickle cell gene, any one of their children have a 25% of developing SCD. If only one parent has the disease, their children have a 50% chance of receiving the gene, but may never have the characteristics of the disease. The Center for Disease Control and Prevention (CDC, 2015), estimates that 90,000

This paper outlines the etiology of sickle cell anemia and the nutrition considerations that is needed to maintain this disease. It also provides a five day menu plan that will greatly provide nutrition needs to prevent having a sickle cell crisis.

Imagine living a life with misleading information about a younger sibling's death. Many wonder why God took them, but not myself. For the simple fact the morning of Madison’s passing God told me everything would be okay. Experiencing this at such young age makes thou think of the extreme; however, it makes thee stronger. The idea of this paper is to break down the information and show everyone how Sepsis breaks down the body. Along, with linking Sepsis with Sickle Cell Trait when the body is already weak and cannot fight off the infection.

Sickle cell anemia is one of the disease out there in the world that cannot be cured. The only thing people suffering from sickle cell can do is get treatments for the symptoms they are suffering from. Not only are there treatments, but there are also things that can be done that can prevent some of the symptoms. Some of the treatments available are different kinds of medications, vaccines, and experimental treatments. When a child suffers from sickle anemia their life could be put on line when catching an infection that may not be life threating to some who may not have that disease, so it is important for children to take antibiotics. From the antibiotics a person can take painkillers to relieve the suffer pain. A drug that is

Madsen et al. developed this classification to asthmatic, cardiac, lymphatic, and sickle cell-related disease states(10, 13, 14). Madsen et al.(10, 13) defined this classification depending on the analysis of prior case studies. Cardiac patients mentioned by them consist of eight Fontan patients, one Glenn shunt patient and three Blalock–Taussig shunt patients. The casts can be immediately expectorated and sometimes patients will cough up large amount from their tracheobronchial tree(14). If the patients do not expectorate, it may delay the diagnosis(14). Patients often need bronchoscopy for the expulsion of cast(14). It is a rare condition that is mentioned in asthma and sickle cell disease patients and has higher mortality in patients with congenital heart disease(10, 13) and death is not unusual due to the occlusion of airway from a cast(14). The pathophysiology and management of plastic bronchitis is not clear due to insufficient clinical data. Over the years, plastic bronchitis has been labeled by various names as Hoffman's bronchitis, cast bronchitis, fibrinous

In this essay I will be talking about sickle cell anemia. Well I was diagnosed with the disease at birth. I have what they call SS, which means I have the full illness. The reason I do is because both of my parents have a trait. This is my story.

It is interesting to know the relationship between race and body structure, skin color, other visible characteristics, enzymatic and genetic variations, susceptibility to diseases and nutritional preferences varies among certain racial groups (Giger, 2013). There are marked intra individual and inter individual inconsistency in SCD. Medical heterogeneity of the disease has been clarified by both genetic and environmental factors. Identified genetic causes adding to differences in clinical severity of the disease include the pattern of sickle cell inheritance. Other modulators of the disease include environmental factors such as access to optimal health care, ambient living conditions, and availability of finance. Proper training of healthcare