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How Osteosarcoma Is Ignited?

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during adolescent growth, which could be why 80% of all osteosarcoma childhood cases occur in the lower long bones and at the time of puberty (Mirabello & Savage 5). The chances of a cell dividing uncontrollably are increased at a time of rapid cell growth when there is many more cells dividing that could go suddenly go uncontrolled, and therefore the answer of how osteosarcoma is ignited could lie in puberty. On the contrary, the answer may be found in a small gene that prevents osteosarcoma patients from responding well to the chemotherapy treatment. Chelsea received chemotherapy through her port, which is a small disc inserted into the chest area that connects to a large vein and serves as a substitute to using an IV through the veins. Most chemotherapy patients have a port put it so that there is minimal damage to the veins in the arms due to the large amount of times it needs to be used. Through this port she received very strong chemotherapy drugs such as cicplatin and methotrexate for about a year that was expected to put her in remission. The doctors told us that her necrosis rate was expected to be about 70%, necrosis rate meaning well the chemotherapy worked, 100% meaning that the tumor is gone, but her necrosis rate didn’t make the 70% mark. In fact, her necrosis rate was zero. The year of suffering through that chemotherapy didn’t help at all, so the doctors had her genome typed out showing all her genes and where mutations had occurred. Chelsea had several

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