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Q: B C D
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why should triacylgycerol be insignificant component of plasma membeane
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- What would happen to the patient if the serum cholinesterase activity were only 10 units of activity per liter rather than the normal activity of about 80 units?Individuals with hyperammonemia are given a-ketoacids as a treatment. Explain.In healthy adults, the concentration of glucose in blood is approximately80 to 110 milligrams per deciliter (mg/dl). After a carbohydrate-richmeal, however, the concentration may spike to 140 mg/dl. Describe thehormonal action that returns blood glucose to normal.
- Why is D-glucose (dextrose), given intravenously to patients rather than sucrose?Glucose has been found to react nonenzymatically with hemoglobin, through Schiff base formation between C-1 of glucose and the amino termini of the β chains. How might this finding be applied in monitoring diabetic patients?Nonalcoholic fatty liver disease is thought to be exacerbated by a diet heavy in sugar. Explain?
- Why do we see lipid octade on renal cell carcinoma amd what are the molecular pathways driving it? Also why is there increase of alkaline phosphatese in some areas?why is chymotrypsin inactive at high pH?In 2-page worth of words (around 500), discuss CYCLAMATE's regulation, allowable levels, and what group of people are at high risk for the side effects of CYCLAMATE in the body.
- One of the following glycosaminoglycans is anti-coagulant O Heparan sulphate O Hyaluronic acid O Heparin sulphate O Keratan sulphate O Chondroitin sulfate searchMany patients with glucose 6- phosphatase deficiency have high serum levels of urate. Hyperuricemia can be induced in normal people by the ingestion of alcohol or by strenuous exercise. Propose a common mechanism that accounts for these findings.Hypercholesterolemic individuals taking statins are sometimes advised to take supplements of coenzyme Q. Explain.