Microbiology With Diseases By Taxonomy (6th Edition)
6th Edition
ISBN: 9780134832302
Author: Robert W. Bauman Ph.D.
Publisher: PEARSON
expand_more
expand_more
format_list_bulleted
Question
Chapter 21, Problem 4CT
Summary Introduction
To explain:
The reason behind the development of ulcers by a mutant strain of Helicobacter pylori, which is urease negative only in patients who either consume antacids in larger amounts or those taking drugs to block acid production.
Introduction:
Scientists have discovered a mutant strain of Helicobacter pylori, which is urease negative. The bacteria of this strain are able to cause infection only in patients who either consume antacids in huge amounts or produce less acid.
Expert Solution & Answer
Want to see the full answer?
Check out a sample textbook solutionStudents have asked these similar questions
All of the following are true about Clostridium tetani, thecausative agent of tetanus, EXCEPT:(a) They are spore-forming rods.(b) They are part of the normal bowel microflora of horses,cattle, and humans.(c) They produce a powerful exotoxin that mediates thedisease.(d) Their toxin elicits muscle stiffness, spasms, and paralysisin the victim.(e) They require oxygen to survive.
Why does Helicobacter pylori cause ulceractions in the lining of the Stomach?
1)The microbes secrete a toxin that prevents the host from detecting invasion of the microbe and thus allows it to thrive in the stomach and damage the host cells.
2)After invasion of the microbe in the cells lining the stomach, they are no longer able to produce mucous to protect the host cells from acid erosion
3)The microbe produces an enzyme that causes the cell to produce potent digestive enzymes that erode the stomach lining.
4)Once the microbe has entered the stomach, it releases digestive enzymes that degrade the mucous secreting cells.
The general public takes for granted the link between an infectious agent and a given infection
Discuss the requirements established by Koch, the nineteenth-century German scientist, before ascertaining, for instance, that Helicobacter pylori bacterium is the cause of the gastric ulcer. Why is it so essential to meet these conditions?
Chapter 21 Solutions
Microbiology With Diseases By Taxonomy (6th Edition)
Ch. 21 - Why do most cases of rickettsial diseases occur...Ch. 21 - Prob. 2TMWCh. 21 - Prob. 3TMWCh. 21 - Why is a cholera pandemic unlikely to become...Ch. 21 - Two patientsa woman and her husband, ages 23 and...Ch. 21 - Most human infections caused by species of...Ch. 21 - Prob. 2MCCh. 21 - Prob. 3MCCh. 21 - Prob. 4MCCh. 21 - The most commonly reported sexually transmitted...
Ch. 21 - Prob. 6MCCh. 21 - Prob. 7MCCh. 21 - Prob. 8MCCh. 21 - Prob. 9MCCh. 21 - Prob. 10MCCh. 21 - Two weeks after a backpacking trip in Tennessee, a...Ch. 21 - Prob. 12MCCh. 21 - Prob. 13MCCh. 21 - Prob. 1VICh. 21 - Prob. 2VICh. 21 - Prob. 1SACh. 21 - Describe the three developmental stages of the...Ch. 21 - Prob. 3SACh. 21 - Prob. 4SACh. 21 - Prob. 5SACh. 21 - Beginning with the ingestion of water contaminated...Ch. 21 - Prob. 1MCh. 21 - Prob. 2MCh. 21 - Prob. 3MCh. 21 - Prob. 4MCh. 21 - Prob. 1CTCh. 21 - Prob. 2CTCh. 21 - Prob. 3CTCh. 21 - Prob. 4CTCh. 21 - Thirty-nine members of an extended family sought...
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- How could an infection with H. pylori be diagnosed given the knowledge that the urease enzyme it produces in the stomach converts urea into ammonia and ultimately into carbon dioxide?arrow_forwardIf there is a causal relationship between the presence of H. pylori and ulcers, how might you explain that 50% of the patients with a normal endoscopic examination were infected with the bacteria?arrow_forwardWhat drugs would you recommend for Tetanus treatment? Explain the mechanism of action of the toxin from Clostridium botulinum. How this mechanism can be used for clinical and cosmetic purposes, including treatment of facial wrinkles?arrow_forward
- Write a paragraph describing the microscopic morphology of: a) Clostridium perfringes b) Clostridium tetani C) Clostridium botulinumarrow_forwardExplain how the body (including cells, organs, organ systems) is affected by the bacterium called Neisseria meningitidis? Are there any long-term effects caused by the bacterium, even after recovery?arrow_forwardWhy is Salmonella typhi a gram negative bacilli? Explain.arrow_forward
- While it is true that not all coliforms are pathogenic under normal conditions, please name two situations that can occur due to infection by pathogenic strains.arrow_forwardWhat are the common pathogens isolated from stool samples? What is the difference between a coliform bacterium and a noncoliform enteric bacterium? What diagnostic test differentiates Proteus and Providencia species from other Enterobacteriaceae? How would you differentiate between serotypes of E. coli? Are the gram-negative enteric bacilli fastidious organisms? Would they survive well outside of the body? If so, what significance would this have in their transmission? Why is serotyping particularly important in Salmonella infections and typhoid fever?arrow_forwardDescribe the microscopic morphology of a) Clostridium perfringes b) Clostridium tetani C) Clostridium botulinumarrow_forward
- In bone marrow culture, immobile, non-hemolyzed, oxidase, catalase, and urease positive gram negative cocobacillar were detected. Which of the following is the most likely factor in this patient?a) Legionella pneumophilab) Brucella mellitensisc) Bordetella bronchisepticad) Salmonella typhimuriume) Stenotrophomonas maltophiliaarrow_forwardCompare the mechanism of virulence for Vibrio cholerae and enterotoxigenic E.coli. Specifically, how do these bacteria bind to host cells and what factors are involved in producing the diarrhea observed in infected patients?arrow_forwardExplain the role of pylori in the pathogenesis of peptic ulcers. Which populations are most susceptible to peptic ulcers? Is H. pylori normal flora? If so, what causes the species to become pathogenic? Construct at table to compare and contrast the 5 steps of microbial pathogenesis. How does extracellular growth differ from intracellular growth? Diagram the 5 step pathogenesis cycle for coli O157:H7, an extracellular, intestinal pathogen acquired by consuming contaminated food/water. Be sure to include the role of exoenzymes and the Shiga exotoxin in your diagram. (2 points) Explain the pathogenesis of Listeria monocytogenes. Be sure to include temperature regulation, intracellular growth, and at risk groups in your discussion.arrow_forward
arrow_back_ios
arrow_forward_ios
Recommended textbooks for you
- Essentials of Pharmacology for Health ProfessionsNursingISBN:9781305441620Author:WOODROWPublisher:Cengage
Essentials of Pharmacology for Health Professions
Nursing
ISBN:9781305441620
Author:WOODROW
Publisher:Cengage
The Human Reproductive System; Author: Professor Dave Explains;https://www.youtube.com/watch?v=TucxiIB76bo;License: Standard YouTube License, CC-BY